Cellular origins of type IV collagen networks in developing glomeruli. - Test2 - elhamkhani - TriplyDB

Cellular origins of type IV collagen networks in developing glomeruli.

Cellular origins of type IV collagen networks in developing glomeruli.

http://www.wikidata.org/entity/Q37259286

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The FOXD1 lineage of kidney perivascular cells and myofibroblasts: functions and responses to injury Role of the podocyte (and glomerular endothelium) in building the GBM Translating genetic findings in hereditary nephrotic syndrome: the missing loops Three-dimensional electron microscopy reveals the evolution of glomerular barrier injury.Cellular and molecular mechanisms in kidney fibrosis.A mouse Col4a4 mutation causing Alport glomerulosclerosis with abnormal collagen α3α4α5(IV) trimers.Deletion of von Hippel-Lindau in glomerular podocytes results in glomerular basement membrane thickening, ectopic subepithelial deposition of collagen {alpha}1{alpha}2{alpha}1(IV), expression of neuroglobin, and proteinuria.Transgenic expression of human LAMA5 suppresses murine Lama5 mRNA and laminin α5 protein deposition.The glomerular basement membrane as a barrier to albumin.Mild electrical stimulation and heat shock ameliorates progressive proteinuria and renal inflammation in mouse model of Alport syndrome Identification of the NC1 domain of {alpha}3 chain as critical for {alpha}3{alpha}4{alpha}5 type IV collagen network assembly.Searching for a treatment for Alport syndrome using mouse models Upregulated expression of integrin α1 in mesangial cells and integrin α3 and vimentin in podocytes of Col4a3-null (Alport) mice Basement membranes: cell scaffoldings and signaling platforms.Lack of collagen XVIII long isoforms affects kidney podocytes, whereas the short form is needed in the proximal tubular basement membrane.Frequency of COL4A3/COL4A4 mutations amongst families segregating glomerular microscopic hematuria and evidence for activation of the unfolded protein response. Focal and segmental glomerulosclerosis is a frequent development during ageing Angiopoietins modulate endothelial adaptation, glomerular and podocyte hypertrophy after uninephrectomy.Identification of microRNAs and their target genes in Alport syndrome using deep sequencing of iPSCs samples.Goodpasture's disease: molecular architecture of the autoantigen provides clues to etiology and pathogenesis.Review series: The cell biology of renal filtration.Mechanisms and consequences of TGF-ß overexpression by podocytes in progressive podocyte disease.An active role for basement membrane assembly and modification in tissue sculpting Murine membranous nephropathy: immunization with α3(IV) collagen fragment induces subepithelial immune complexes and FcγR-independent nephrotic syndrome.COL4A2 mutation associated with familial porencephaly and small-vessel disease.X-Linked Alport Dogs Demonstrate Mesangial Filopodial Invasion of the Capillary Tuft as an Early Event in Glomerular Damage.Glomerular basement membrane and related glomerular disease ER stress and basement membrane defects combine to cause glomerular and tubular renal disease resulting from Col4a1 mutations in mice.Tensin2-deficient mice on FVB/N background develop severe glomerular disease.Laminin and type IV collagen isoform substitutions occur in temporally and spatially distinct patterns in developing kidney glomerular basement membranes.Nanoscale protein architecture of the kidney glomerular basement membrane.Diabetes-Induced Reactive Oxygen Species: Mechanism of Their Generation and Role in Renal Injury Feasibility of repairing glomerular basement membrane defects in Alport syndrome.Glomerular cell cross-talk influences composition and assembly of extracellular matrix Glomerular basement membrane composition and the filtration barrier.Organogenesis of the kidney glomerulus: focus on the glomerular basement membrane.Advances in human antiglomerular basement membrane disease.The glomerular basement membrane.An update on the pathomechanisms and future therapies of Alport syndrome.Alport syndrome--insights from basic and clinical research.Cell-matrix adhesion of podocytes in physiology and disease.

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Cellular origins of type IV collagen networks in developing glomeruli.

http://www.wikidata.org/entity/Q37259286

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 07 May 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cellular origins of type IV collagen networks in developing glomeruli.

@en

Cellular origins of type IV collagen networks in developing glomeruli.

@nl

type

label

Cellular origins of type IV collagen networks in developing glomeruli.

@en

Cellular origins of type IV collagen networks in developing glomeruli.

@nl

prefLabel

Cellular origins of type IV collagen networks in developing glomeruli.

@en

Cellular origins of type IV collagen networks in developing glomeruli.

@nl

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Journal of the American Society of Nephrology

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Cellular origins of type IV collagen networks in developing glomeruli

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Dale R Abrahamson

http://www.w3.org/2001/XMLSchema#string

Larysa Stroganova

http://www.w3.org/2001/XMLSchema#string

Patricia L St John

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P2860

Mammalian collagen IV

Human laminin beta2 deficiency causes congenital nephrosis with mesangial sclerosis and distinct eye abnormalities

Insertional mutation of the collagen genes Col4a3 and Col4a4 in a mouse model of Alport syndrome

The renal glomerulus of mice lacking s-laminin/laminin beta 2: nephrosis despite molecular compensation by laminin beta 1

Promoter traps in embryonic stem cells: a genetic screen to identify and mutate developmental genes in mice

Renal basement membrane components.

Bone-marrow-derived stem cells repair basement membrane collagen defects and reverse genetic kidney disease.

Alport's syndrome, Goodpasture's syndrome, and type IV collagen.

Glomerular expression of type IV collagen chains in normal and X-linked Alport syndrome kidneys

A human-mouse chimera of the alpha3alpha4alpha5(IV) collagen protomer rescues the renal phenotype in Col4a3-/- Alport mice.

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1471-1479

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scholarly article

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10.1681/ASN.2008101086

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7

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20

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Dorin-Bogdan Borza

Billy G. Hudson

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2009-05-07T00:00:00Z

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19423686

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2709682

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