IL2Rβ-dependent signals drive terminal exhaustion and suppress memory development during chronic viral infection. - Test2 - elhamkhani - TriplyDB

IL2Rβ-dependent signals drive terminal exhaustion and suppress memory development during chronic viral infection.

IL2Rβ-dependent signals drive terminal exhaustion and suppress memory development during chronic viral infection.

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IL2Rβ-dependent signals drive terminal exhaustion and suppress memory development during chronic viral infection.

http://www.wikidata.org/entity/Q37264094

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 29 August 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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IL2Rβ-dependent signals drive ...... uring chronic viral infection.

@en

IL2Rβ-dependent signals drive ...... uring chronic viral infection.

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IL2Rβ-dependent signals drive ...... uring chronic viral infection.

@en

IL2Rβ-dependent signals drive ...... uring chronic viral infection.

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IL2Rβ-dependent signals drive ...... uring chronic viral infection.

@en

IL2Rβ-dependent signals drive ...... uring chronic viral infection.

@nl

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PNAS.1604256113

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Proceedings of the National Academy of Sciences of the United States of America

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IL2Rβ-dependent signals drive ...... uring chronic viral infection.

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Eva Michaud

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Ines Boufaied

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Jean-Christophe Beltra

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Julie Bruneau

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Moana Boulangé

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Naglaa H Shoukry

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Nathalie Bédard

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Sara Bourbonnais

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Tania Charpentier

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P2860

Mechanistic and structural insight into the functional dichotomy between IL-2 and IL-15

Molecular and cellular insights into T cell exhaustion

Restoring function in exhausted CD8 T cells during chronic viral infection

Bcl6 acts as an amplifier for the generation and proliferative capacity of central memory CD8+ T cells

CD4+ T cell regulation of CD25 expression controls development of short-lived effector CD8+ T cells in primary and secondary responses.

Intrinsic functional dysregulation of CD4 T cells occurs rapidly following persistent viral infection

Interleukin-7 mediates the homeostasis of naïve and memory CD8 T cells in vivo.

Gamma(c) deficiency precludes CD8+ T cell memory despite formation of potent T cell effectors.

Interleukin-2 and inflammation induce distinct transcriptional programs that promote the differentiation of effector cytolytic T cells.

Cooperation of Tim-3 and PD-1 in CD8 T-cell exhaustion during chronic viral infection.

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E5444-53

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scholarly article

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10.1073/PNAS.1604256113

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37

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113

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Hélène Decaluwe

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2016-08-29T00:00:00Z

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27573835

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5027416

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