Multiple system atrophy: a primary oligodendrogliopathy.
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Review: Multiple system atrophy: emerging targets for interventional therapiesNeuroinflammation in Multiple System Atrophy: Response to and Cause of α-Synuclein AggregationGlia and alpha-synuclein in neurodegeneration: A complex interactionExploring myelin dysfunction in multiple system atrophyMultiple sclerosis: molecular mechanisms and therapeutic opportunitiesTubulin polymerization-promoting protein (TPPP/p25) is critical for oligodendrocyte differentiationGenomic and proteomic responses to environmentally relevant exposures to dieldrin: indicators of neurodegeneration?HDAC6 inhibition results in tau acetylation and modulates tau phosphorylation and degradation in oligodendrocytesFragile X tremor ataxia syndrome and white matter dementia.Defective adult oligodendrocyte and Schwann cell development, pigment pattern, and craniofacial morphology in puma mutant zebrafish having an alpha tubulin mutation.Systemic proteasome inhibition triggers neurodegeneration in a transgenic mouse model expressing human α-synuclein under oligodendrocyte promoter: implications for multiple system atrophy.Distinct Parameters in the EEG of the PLP α-SYN Mouse Model for Multiple System Atrophy Reinforce Face ValidityDifferential deployment of REST and CoREST promotes glial subtype specification and oligodendrocyte lineage maturation.Behavioral and neurophysiological effects of transdermal rotigotine in atypical parkinsonism.Rotigotine is safe and efficacious in Atypical Parkinsonism Syndromes induced by both α-synucleinopathy and tauopathy.Towards translational therapies for multiple system atrophyNovel oligodendroglial alpha synuclein viral vector models of multiple system atrophy: studies in rodents and nonhuman primates.Environmentally relevant exposure to 17alpha-ethinylestradiol affects the telencephalic proteome of male fathead minnowsAn investigation of diffusion imaging techniques in the evaluation of spinocerebellar ataxia and multisystem atrophy.Mitochondrial inhibitor 3-nitroproprionic acid enhances oxidative modification of alpha-synuclein in a transgenic mouse model of multiple system atrophyMesenchymal stem cells in a transgenic mouse model of multiple system atrophy: immunomodulation and neuroprotectionMultiple system atrophy as emerging template for accelerated drug discovery in α-synucleinopathiesThe neuropathology, pathophysiology and genetics of multiple system atrophy.Relocation of p25α/tubulin polymerization promoting protein from the nucleus to the perinuclear cytoplasm in the oligodendroglia of sporadic and COQ2 mutant multiple system atrophy.Differential involvement of the periaqueductal gray in multiple system atrophy.SHC2 gene copy number in multiple system atrophy (MSA)Multiple system atrophy: current and future approaches to managementIncreased neuronal α-synuclein pathology associates with its accumulation in oligodendrocytes in mice modeling α-synucleinopathiesModelling progressive autonomic failure in MSA: where are we now?Genetic players in multiple system atrophy: unfolding the nature of the beast.The brainstem pathologies of Parkinson's disease and dementia with Lewy bodies.The role of environmental exposures in neurodegeneration and neurodegenerative diseases.Filamentous aggregations of phosphorylated α-synuclein in Schwann cells (Schwann cell cytoplasmic inclusions) in multiple system atrophyParkinson's disease induced pluripotent stem cells with triplication of the α-synuclein locus.Non-gaussianity of low frequency heart rate variability and sympathetic activation: lack of increases in multiple system atrophy and Parkinson diseaseOverexpression of α-synuclein in oligodendrocytes does not increase susceptibility to focal striatal excitotoxicity.Changes in the miRNA-mRNA Regulatory Network Precede Motor Symptoms in a Mouse Model of Multiple System Atrophy: Clinical ImplicationsProgressive retinal structure abnormalities in multiple system atrophy.Suppression of dynamin GTPase decreases α-synuclein uptake by neuronal and oligodendroglial cells: a potent therapeutic target for synucleinopathy.Glucocerebrosidase mutations in diffuse Lewy body disease
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Multiple system atrophy: a primary oligodendrogliopathy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on September 2008
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Multiple system atrophy: a primary oligodendrogliopathy.
@en
Multiple system atrophy: a primary oligodendrogliopathy.
@nl
type
label
Multiple system atrophy: a primary oligodendrogliopathy.
@en
Multiple system atrophy: a primary oligodendrogliopathy.
@nl
prefLabel
Multiple system atrophy: a primary oligodendrogliopathy.
@en
Multiple system atrophy: a primary oligodendrogliopathy.
@nl
P2860
P50
P356
P1433
P1476
Multiple system atrophy: a primary oligodendrogliopathy
@en
P2093
Kurt A Jellinger
Werner Poewe
P2860
P304
P356
10.1002/ANA.21465
P577
2008-09-01T00:00:00Z