Embryonic type Na+ channel β-subunit, SCN3B masks the disease phenotype of Brugada syndrome.
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Modeling Inherited Arrhythmia Disorders Using Induced Pluripotent Stem Cell-Derived Cardiomyocytes.From Bench to Market: Preparing Human Pluripotent Stem Cells Derived Cardiomyocytes for Various ApplicationsEmerin plays a crucial role in nuclear invagination and in the nuclear calcium transientFrom iPSC towards cardiac tissue-a road under construction.Human heart disease: lessons from human pluripotent stem cell-derived cardiomyocytes.Mechanisms of noncovalent β subunit regulation of NaV channel gating.Flecainide ameliorates arrhythmogenicity through NCX flux in Andersen-Tawil syndrome-iPS cell-derived cardiomyocytes.Mechanisms and models of cardiac sodium channel inactivation.Using fluorescence to understand β subunit-NaV channel interactions.
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Embryonic type Na+ channel β-subunit, SCN3B masks the disease phenotype of Brugada syndrome.
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 28 September 2016
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@en
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
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type
label
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@en
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@nl
prefLabel
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@en
Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@nl
P2093
P2860
P50
P356
P1433
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Embryonic type Na+ channel β-s ...... phenotype of Brugada syndrome.
@en
P2093
Atsushi Tanaka
Chikaaki Motoda
Gakuto Yozu
Hisayuki Hashimoto
Kaichiro Kamiya
Keiichi Fukuda
Masaya Shimojima
Minoru Horie
Mitsushige Murata
P2860
P2888
P356
10.1038/SREP34198
P407
P577
2016-09-28T00:00:00Z