Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs. - Test2 - elhamkhani - TriplyDB

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

http://www.wikidata.org/entity/Q37295712

about

Importance of metabolic rate to the relationship between the number of genes in a functional category and body size in Peto's paradox for cancer Foot-and-mouth disease virus infection inhibits LGP2 protein expression to exaggerate inflammatory response and promote viral replication.The intersection of radiotherapy and immunotherapy: mechanisms and clinical implications.Promising Targets for Cancer Immunotherapy: TLRs, RLRs, and STING-Mediated Innate Immune Pathways Towards precision radiotherapy for use with immune checkpoint blockers.Crosstalk between Cytoplasmic RIG-I and STING Sensing Pathways.RIG-I: a multifunctional protein beyond a pattern recognition receptor.Mechanisms regulating immune surveillance of cellular stress in cancer.Suppression of Transposable Elements in Leukemic Stem Cells.Innate sensing of cancer's non-immunologic hallmarks.Stimulating Innate Immunity to Enhance Radiation Therapy-Induced Tumor Control.Cell death and immunity in cancer: From danger signals to mimicry of pathogen defense responses.Host STING-dependent MDSC mobilization drives extrinsic radiation resistance.PARP1 depletion induces RIG-I-dependent signaling in human cancer cells.Targeting Cytosolic Nucleic Acid-Sensing Pathways for Cancer Immunotherapies.A GTPase-activating protein binding protein (G3BP1) / antiviral protein relay conveys arteriosclerotic Wnt signals in aortic smooth muscle cells.Effects of retinoic acid-inducible gene-I-like receptors activations and ionizing radiation cotreatment on cytotoxicity against human non-small cell lung cancer in vitro.Harnessing RIG-I and intrinsic immunity in the tumor microenvironment for therapeutic cancer treatment.The DNA damage response in immunotherapy and radiation

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Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

http://www.wikidata.org/entity/Q37295712

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 28 March 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@en

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@nl

type

label

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@en

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@nl

prefLabel

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@en

Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

@nl

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ONCOTARGET.8420

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Cancer therapies activate RIG-I-like receptor pathway through endogenous non-coding RNAs.

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Akash D Parekh

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Anthony C Wong

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Diana Rose E Ranoa

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Jonathan P Staley

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Jorge Andrade

http://www.w3.org/2001/XMLSchema#string

Lei Huang

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Nikolai N Khodarev

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Sean P Pitroda

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Shizuo Akira

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Takashi Satoh

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P2860

Ultrafast and memory-efficient alignment of short DNA sequences to the human genome

Immune signaling by RIG-I-like receptors

Recognition of 5' triphosphate by RIG-I helicase requires short blunt double-stranded RNA as contained in panhandle of negative-strand virus.

Significance analysis of microarrays applied to the ionizing radiation response

GENCODE: the reference human genome annotation for The ENCODE Project

DICER1 loss and Alu RNA induce age-related macular degeneration via the NLRP3 inflammasome and MyD88

A diverse range of gene products are effectors of the type I interferon antiviral response

RNA- and virus-independent inhibition of antiviral signaling by RNA helicase LGP2

Prion-like polymerization as a signaling mechanism

Control of adaptive immunity by the innate immune system

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26496-26515

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scholarly article

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10.18632/ONCOTARGET.8420

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18

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Ralph Weichselbaum

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2016-03-28T00:00:00Z

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299458209

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5041995

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