Generation of highly-reactive oxygen species is closely related to hair cell damage in rat organ of Corti treated with gentamicin.
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New developments in aminoglycoside therapy and ototoxicityWnt activation protects against neomycin-induced hair cell damage in the mouse cochleaEffects of NSAIDs on the Inner Ear: Possible Involvement in Cochlear Protection.Mitochondrial peroxiredoxin 3 regulates sensory cell survival in the cochleaReduced TRMU expression increases the sensitivity of hair-cell-like HEI-OC-1 cells to neomycin damage in vitroBmi1 regulates auditory hair cell survival by maintaining redox balance.Assessment of nutrient supplement to reduce gentamicin-induced ototoxicity.Mechanisms of sensorineural cell damage, death and survival in the cochlea.Molecular regulation of auditory hair cell death and approaches to protect sensory receptor cells and/or stimulate repair following acoustic trauma.Adjudin protects rodent cochlear hair cells against gentamicin ototoxicity via the SIRT3-ROS pathway.Extracellular gentamicin reduces the activity of connexin hemichannels and interferes with purinergic Ca(2+) signaling in HeLa cells.Efficacy of three drugs for protecting against gentamicin-induced hair cell and hearing losses.Cytomegalovirus-induced sensorineural hearing loss with persistent cochlear inflammation in neonatal miceMitochondrial calcium uptake underlies ROS generation during aminoglycoside-induced hair cell deathc-Myb knockdown increases the neomycin-induced damage to hair-cell-like HEI-OC1 cells in vitroKorean red ginseng prevents gentamicin-induced hearing loss in rats.Profiling drug-induced cell death pathways in the zebrafish lateral line.NaHS Protects Cochlear Hair Cells from Gentamicin-Induced Ototoxicity by Inhibiting the Mitochondrial Apoptosis PathwayActivation of apoptotic pathways in the absence of cell death in an inner-ear immortomouse cell lined-Methionine reduces tobramycin-induced ototoxicity without antimicrobial interference in animal models.Morin hydrate promotes inner ear neural stem cell survival and differentiation and protects cochlea against neuronal hearing loss.Inhibition of ARC decreases the survival of HEI-OC-1 cells after neomycin damage in vitroFenofibrate exerts protective effects against gentamicin-induced toxicity in cochlear hair cells by activating antioxidant enzymes.Analysis of frequency loss as a prognostic factor in idiopathic sensorineural hearing loss.Intracochlear electrical stimulation suppresses apoptotic signaling in rat spiral ganglion neurons after deafening in vivo.An Antioxidant Screen Identifies Candidates for Protection of Cochlear Hair Cells from Gentamicin ToxicityCochlear pharmacokinetics of cisplatin: an in vivo study in the guinea pig.A kinase inhibitor library screen identifies novel enzymes involved in ototoxic damage to the murine organ of CortiPI3K and Inhibitor of Apoptosis Proteins Modulate Gentamicin- Induced Hair Cell Death in the Zebrafish Lateral Line.Efficacy of 3 different steroid treatments for sudden sensorineural hearing loss: a prospective, randomized trial.Autophagy protects auditory hair cells against neomycin-induced damage.The efficiency of intratympanic dexamethasone injection as a sequential treatment after initial systemic steroid therapy for sudden sensorineural hearing loss.Meclofenamic Acid Reduces Reactive Oxygen Species Accumulation and Apoptosis, Inhibits Excessive Autophagy, and Protects Hair Cell-Like HEI-OC1 Cells From Cisplatin-Induced Damage.STK33 alleviates gentamicin-induced ototoxicity in cochlear hair cells and House Ear Institute-Organ of Corti 1 cells
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Generation of highly-reactive oxygen species is closely related to hair cell damage in rat organ of Corti treated with gentamicin.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 24 March 2009
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Generation of highly-reactive ...... Corti treated with gentamicin.
@en
Generation of highly-reactive ...... Corti treated with gentamicin.
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type
label
Generation of highly-reactive ...... Corti treated with gentamicin.
@en
Generation of highly-reactive ...... Corti treated with gentamicin.
@nl
prefLabel
Generation of highly-reactive ...... Corti treated with gentamicin.
@en
Generation of highly-reactive ...... Corti treated with gentamicin.
@nl
P2093
P2860
P1433
P1476
Generation of highly-reactive ...... Corti treated with gentamicin.
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P2093
P2860
P304
P356
10.1016/J.NEUROSCIENCE.2009.02.085
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P577
2009-03-24T00:00:00Z