Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia. - Test2 - elhamkhani - TriplyDB

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

http://www.wikidata.org/entity/Q37343021

about

Ikaros: Exploiting and targeting the hematopoietic stem cell niche in B-progenitor acute lymphoblastic leukemia.The making of a lymphocyte: the choice among disparate cell fates and the IKAROS enigma.Transcriptional circuits in B cell transformation.LMO2 is required for TAL1 DNA binding activity and initiation of definitive haematopoiesis at the haemangioblast stage.Transcriptional and Microenvironmental Regulation of Lineage Ambiguity in Leukemia.Regulation of the Hippo Pathway Transcription Factor TEAD.Germline Genetic IKZF1 Variation and Predisposition to Childhood Acute Lymphoblastic Leukemia.YAP and TAZ are dispensable for physiological and malignant haematopoiesis.The many faces of IKZF1 in B-cell precursor acute lymphoblastic leukemia.It is a differentiation game: STAT5 in a new role.

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Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

http://www.wikidata.org/entity/Q37343021

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on September 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@en

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@nl

type

label

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@en

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@nl

prefLabel

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@en

Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

@nl

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Genes & Development

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Superenhancer reprogramming drives a B-cell-epithelial transition and high-risk leukemia.

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Akinola Olumide Emmanuel

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Fotini Gounari

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Ila Joshi

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Julien Fitamant

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Katia Georgopoulos

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Mariko Kashiwagi

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Mikael Sigvardsson

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Nabeel El-Bardeesy

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Nilamani Jena

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Rajesh Somasundaram

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P2860

A large fraction of extragenic RNA pol II transcription sites overlap enhancers

Model-based analysis of ChIP-Seq (MACS)

An essential role for BLNK in human B cell development

Acute lymphoblastic leukaemia

Simple combinations of lineage-determining transcription factors prime cis-regulatory elements required for macrophage and B cell identities

Histone H3K27ac separates active from poised enhancers and predicts developmental state

Fast and accurate short read alignment with Burrows-Wheeler transform

Histone methylation by PRC2 is inhibited by active chromatin marks

IKZF1 (Ikaros) deletions in BCR-ABL1-positive acute lymphoblastic leukemia are associated with short disease-free survival and high rate of cumulative incidence of relapse: a GIMEMA AL WP report.

IKZF1 deletions predict relapse in uniformly treated pediatric precursor B-ALL.

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1971-1990

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10.1101/GAD.283762.116

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17

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2016-09-01T00:00:00Z

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