Junctional adhesion molecule-C mediates leukocyte infiltration in response to ischemia reperfusion injury. - Test2 - elhamkhani - TriplyDB

Junctional adhesion molecule-C mediates leukocyte infiltration in response to ischemia reperfusion injury.

Junctional adhesion molecule-C mediates leukocyte infiltration in response to ischemia reperfusion injury.

http://www.wikidata.org/entity/Q37353304

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Divergent JAM-C Expression Accelerates Monocyte-Derived Cell Exit from Atherosclerotic Plaques The spatiotemporal localization of JAM-C following sciatic nerve crush in adult rats The junctional adhesion molecule JAM-C regulates polarized transendothelial migration of neutrophils in vivo.Unique cell type-specific junctional complexes in vascular endothelium of human and rat liver sinusoids.Targeting stem cell niches and trafficking for cardiovascular therapy Blocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.Acute kidney injury.Ischemic kidney injury and mechanisms of tissue repair.Schwann cell-specific JAM-C-deficient mice reveal novel expression and functions for JAM-C in peripheral nerves.Adrenergic nerves govern circadian leukocyte recruitment to tissues.Monocyte ADAM17 promotes diapedesis during transendothelial migration: identification of steps and substrates targeted by metalloproteinases Endothelial cell junctional adhesion molecule C plays a key role in the development of tumors in a murine model of ovarian cancer.Recent developments and complexities in neutrophil transmigration Function of junctional adhesion molecules (JAMs) in leukocyte migration and homeostasis.Tight junction dynamics: the role of junctional adhesion molecules (JAMs).JAM-related proteins in mucosal homeostasis and inflammation Identification of novel molecular signatures of IgA nephropathy through an integrative -omics analysis.Role of reverse transendothelial migration of neutrophils in inflammation.Endothelial Cell Junctional Adhesion Molecules: Role and Regulation of Expression in Inflammation.Leukotriene B4-Neutrophil Elastase Axis Drives Neutrophil Reverse Transendothelial Cell Migration In Vivo.Junctional Adhesion Molecules (JAMs): The JAM-Integrin Connection.Ischemia-reperfusion injury in stroke: impact of the brain barriers and brain immune privilege on neutrophil function

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Junctional adhesion molecule-C mediates leukocyte infiltration in response to ischemia reperfusion injury.

http://www.wikidata.org/entity/Q37353304

description

article científic

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article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

@tr

scientific article published on 02 July 2009

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

@cs

name

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@en

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@nl

type

label

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@en

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@nl

prefLabel

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@en

Junctional adhesion molecule-C ...... o ischemia reperfusion injury.

@nl

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P356

ATVBAHA.109.187559

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Arteriosclerosis, Thrombosis, and Vascular Biology

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Junctional adhesion molecule-C ...... to ischemia reperfusion injury

@en

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Abigail Woodfin

http://www.w3.org/2001/XMLSchema#string

Alessandra Marrelli

http://www.w3.org/2001/XMLSchema#string

Beat A Imhof

http://www.w3.org/2001/XMLSchema#string

Christoph Thiemermann

http://www.w3.org/2001/XMLSchema#string

Mathieu-Benoit Voisin

http://www.w3.org/2001/XMLSchema#string

Paolo Meda

http://www.w3.org/2001/XMLSchema#string

Sussan Nourshargh

http://www.w3.org/2001/XMLSchema#string

P2860

Cutting edge: combined treatment of TNF-alpha and IFN-gamma causes redistribution of junctional adhesion molecule in human endothelial cells

JAM-C is a component of desmosomes and a ligand for CD11b/CD18-mediated neutrophil transepithelial migration

Antibody against junctional adhesion molecule-C inhibits angiogenesis and tumor growth

The junctional adhesion molecule 3 (JAM-3) on human platelets is a counterreceptor for the leukocyte integrin Mac-1

Leukocyte-endothelial-cell interactions in leukocyte transmigration and the inflammatory response

Cloning of human junctional adhesion molecule 3 (JAM3) and its identification as the JAM2 counter-receptor

Junctional adhesion molecule-2 (JAM-2) promotes lymphocyte transendothelial migration

JAM2 interacts with alpha4beta1. Facilitation by JAM3

Heterogeneity of endothelial junctions is reflected by differential expression and specific subcellular localization of the three JAM family members

Junctional adhesion molecule-C regulates the early influx of leukocytes into tissues during inflammation

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1509-1515

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scholarly article

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10.1161/ATVBAHA.109.187559

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P433

10

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29

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Michel Aurrand-Lions

Costantino Pitzalis

Christoph Scheiermann

Nimesh S A Patel

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2009-07-02T00:00:00Z

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19574560

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reperfusion injury

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2746810

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