Glucose-6-phosphate dehydrogenase contributes to the regulation of glucose uptake in skeletal muscle. - Test2 - elhamkhani - TriplyDB

Glucose-6-phosphate dehydrogenase contributes to the regulation of glucose uptake in skeletal muscle.

Glucose-6-phosphate dehydrogenase contributes to the regulation of glucose uptake in skeletal muscle.

http://www.wikidata.org/entity/Q37368731

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Resistance training regulates gene expression of molecules associated with intramyocellular lipids, glucose signaling and fiber size in old rats.

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Glucose-6-phosphate dehydrogenase contributes to the regulation of glucose uptake in skeletal muscle.

http://www.wikidata.org/entity/Q37368731

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 09 September 2016

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

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Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

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type

label

Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

@en

Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

@nl

prefLabel

Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

@en

Glucose-6-phosphate dehydrogen ...... ose uptake in skeletal muscle.

@nl

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J.MOLMET.2016.09.002

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Molecular Metabolism

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Glucose-6-phosphate dehydrogen ...... cose uptake in skeletal muscle

@en

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Borivoj Zivanovic

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Bronwyn A Kingwell

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Clinton R Bruce

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Darren C Henstridge

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Dorit Samocha-Bonet

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Gordon S Lynch

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Grant R Drummond

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Mark A Febbraio

http://www.w3.org/2001/XMLSchema#string

Peter Iliades

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Robert S Lee-Young

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P2860

Metformin Increases AMP-Activated Protein Kinase Activity in Skeletal Muscle of Subjects With Type 2 Diabetes

Insulin increases cell surface GLUT4 levels by dose dependently discharging GLUT4 into a cell surface recycling pathway

Insulin resistance is a cellular antioxidant defense mechanism

Glucose-6-phosphate dehydrogenase: a "housekeeping" enzyme subject to tissue-specific regulation by hormones, nutrients, and oxidant stress

Hyperglycemia inhibits endothelial nitric oxide synthase activity by posttranslational modification at the Akt site

Increased oxidative stress in obesity and its impact on metabolic syndrome

Pathogenesis of insulin resistance in skeletal muscle.

Overexpression of glucose-6-phosphate dehydrogenase is associated with lipid dysregulation and insulin resistance in obesity

Endothelial nitric oxide synthase is central to skeletal muscle metabolic regulation and enzymatic signaling during exercise in vivo.

High glucose inhibits glucose-6-phosphate dehydrogenase, leading to increased oxidative stress and beta-cell apoptosis.

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1083-1091

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scholarly article

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10.1016/J.MOLMET.2016.09.002

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11

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5

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Glenn K. McConell

Andrew L. Siebel

Jerry R Greenfield

Michael J Kraakman

Nolan J Hoffman

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2016-09-09T00:00:00Z

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27818934

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