about
A family of cyclin homologs that control the G1 phase in yeastThe BTB-zinc finger transcription factor abrupt acts as an epithelial oncogene in Drosophila melanogaster through maintaining a progenitor-like cell statehScrib is a functional homologue of the Drosophila tumour suppressor ScribbleAPC/CFzr/Cdh1 promotes cell cycle progression during the Drosophila endocycleThe Drosophila Geminin homolog: roles for Geminin in limiting DNA replication, in anaphase and in neurogenesisDifferential regulation of protein tyrosine kinase signaling by Dock and the PTP61F variants.The lethal giant larvae tumour suppressor mutation requires dMyc oncoprotein to promote clonal malignancy.Scribble mutants promote aPKC and JNK-dependent epithelial neoplasia independently of Crumbs.Dominant negative protein kinase mutations that confer a G1 arrest phenotype.lgl Regulates the Hippo Pathway Independently of Fat/Dachs, Kibra/Expanded/Merlin and dRASSF/dSTRIPAK.Loss of the Drosophila cell polarity regulator Scribbled promotes epithelial tissue overgrowth and cooperation with oncogenic Ras-Raf through impaired Hippo pathway signalingDrosophila cyclin E interacts with components of the Brahma complexAbnormalities in cell proliferation and apico-basal cell polarity are separable in Drosophila lgl mutant clones in the developing eye.A genetic screen for dominant modifiers of a cyclin E hypomorphic mutation identifies novel regulators of S-phase entry in Drosophila.Death to flies: Drosophila as a model system to study programmed cell death.Identification of novel Ras-cooperating oncogenes in Drosophila melanogaster: a RhoGEF/Rho-family/JNK pathway is a central driver of tumorigenesis.DRONC, an ecdysone-inducible Drosophila caspaseBuffy, a Drosophila Bcl-2 protein, has anti-apoptotic and cell cycle inhibitory functions.Bcl-2 in cell cycle regulation.BTB-Zinc Finger Oncogenes Are Required for Ras and Notch-Driven Tumorigenesis in Drosophila.Cooperation of the BTB-Zinc finger protein, Abrupt, with cytoskeletal regulators in Drosophila epithelial tumorigenesis.Regulation of Notch signaling and endocytosis by the Lgl neoplastic tumor suppressor.Using Drosophila melanogaster to map human cancer pathways.Debcl, a proapoptotic Bcl-2 homologue, is a component of the Drosophila melanogaster cell death machinery.An in vivo large-scale chemical screening platform using Drosophila for anti-cancer drug discovery.Control of tumourigenesis by the Scribble/Dlg/Lgl polarity module.scribble mutants cooperate with oncogenic Ras or Notch to cause neoplastic overgrowth in Drosophila.The Brm-HDAC3-Erm repressor complex suppresses dedifferentiation in Drosophila type II neuroblast lineages.Upstream regulation of the hippo size control pathway.Lgl/aPKC and Crb regulate the Salvador/Warts/Hippo pathwayWhen cell cycle meets development.The Scribble-Dlg-Lgl polarity module in development and cancer: from flies to man.Hfp inhibits Drosophila myc transcription and cell growth in a TFIIH/Hay-dependent manner.Tissue growth and tumorigenesis in Drosophila: cell polarity and the Hippo pathway.STRICA, a novel Drosophila melanogaster caspase with an unusual serine/threonine-rich prodomain, interacts with DIAP1 and DIAP2.A Kinome RNAi Screen in Drosophila Identifies Novel Genes Interacting with Lgl, aPKC, and Crb Cell Polarity Genes in Epithelial Tissues.Autophagy suppresses Ras-driven epithelial tumourigenesis by limiting the accumulation of reactive oxygen species.A fission yeast B-type cyclin functioning early in the cell cycle.Death takes a holiday--non-apoptotic role for caspases in cell migration and invasion.Drosophila Hfp negatively regulates dmyc and stg to inhibit cell proliferation.
P50
Q24608767-1CDED13E-1F81-43AF-9450-B8323BF50047Q27321236-E06A4BEE-8427-4B6E-9F86-69F150894969Q28235003-49056789-0CF5-4E5F-8778-7F093095816AQ28271653-2271402C-6B5B-4047-8C95-807AF4AEE4F4Q28362218-A003CD53-0423-4785-BFF6-D7D28DDC4021Q30152628-B04E5E92-F160-42CC-BCEE-2DC7A1B64829Q30494710-6417BC6F-EDA4-4E7F-BF91-BEB9D5471B06Q33506529-F35287C5-7C42-4573-9A6B-649E3A8A2471Q33582283-5EF4DA37-4DF6-4BA8-9992-DA6A97397EA6Q33820271-B0579F67-30FA-4ECA-910B-A51A11BE514BQ34034164-C8F5CB1A-F5D3-4359-B4EB-6802DB3976D8Q34089471-F78F54A4-D91F-4501-A2F1-49C45EA50E55Q34283595-3ABDC27B-9A90-42DB-BB97-D41A58703156Q34567130-AF59BA32-F5D7-4C1A-AD9A-F542D70E79BDQ34693186-61945F65-6ED5-4DE8-AD48-2766BB0DC4D4Q35060075-6623F405-E69C-423B-A899-A53D96E077C7Q35124238-3538A5AE-1513-4B00-9396-7340247B4100Q35160263-2187E80E-DCF4-4DAA-8F8D-03A2EA4ABE28Q35601054-B0A4E402-3CEB-449A-BA7D-5445BF390A44Q35711047-0DD3210E-1650-47F2-970C-EDB0D1513CD2Q35973348-7DAF1DBC-46F5-4E00-9BB2-D3E042647F94Q36185779-73FB4A96-EB3A-4AAC-8DA8-3F5D1B057F89Q36202195-3D9157F9-8954-42C3-93F3-3CAFF8D2A811Q36316368-DE260626-E702-47CD-A612-A71E87AE1BC9Q36683116-B7E51F1E-FE24-4CA7-AE37-6D9198906BF7Q37333588-8F8FEDF6-EC39-453B-8409-679BAF760A96Q37367551-0A1B4773-9070-47C5-B40B-8F87287C94CCQ37620996-A7A8CE63-2326-486F-9636-F3FF8DAB5B22Q37771533-258E688C-3C37-4C0D-A3E3-BB626970C9A2Q37783515-E29A9F97-97DD-46CB-8C2D-EACAD60D095EQ37963141-39B59D68-BBA9-447B-A197-DB95B1D51297Q38038640-D2A3070D-90A9-4086-8C15-340EB840EA96Q38342097-BB57A305-064A-43FA-B2C7-597B5166705FQ39212521-F0D27893-0914-420E-956F-109E311BA5F4Q40781431-FC08EFDA-FFE2-41E3-871B-01A509F4F257Q41380844-EB9031CA-C803-4AD5-8677-0C5AAE49095DQ42268242-53E53189-1A79-4922-86EB-A9319C3DFFB0Q42620500-E50CF812-4CC7-4C9A-BE61-4A20540D2133Q43202991-DF01C504-42F0-45D7-B9DB-C77B09CDE630Q44781977-FFAB6570-D4EA-4B61-B0DD-B3126FDDEE42
P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Helena E Richardson
@nl
Helena E Richardson
@sl
Helena E. Richardson
@en
Helena E. Richardson
@es
type
label
Helena E Richardson
@nl
Helena E Richardson
@sl
Helena E. Richardson
@en
Helena E. Richardson
@es
altLabel
H Richardson
@en
Richardson, HE
@en
prefLabel
Helena E Richardson
@nl
Helena E Richardson
@sl
Helena E. Richardson
@en
Helena E. Richardson
@es
P1153
P1053
A-8080-2013
P106
P1153
55230421700
55545096400
7102123652
P21
P31
P3829
P496
0000-0003-3852-4953