about
Fis is essential for capsule production in Pasteurella multocida and regulates expression of other important virulence factorsIn silico prediction of Gallibacterium anatis pan-immunogensAnalysis of the Pasteurella multocida outer membrane sub-proteome and its response to the in vivo environment of the natural host.NetB, a new toxin that is associated with avian necrotic enteritis caused by Clostridium perfringens.Comparative transcriptomic analysis of Porphyromonas gingivalis biofilm and planktonic cells.The Burkholderia pseudomallei type III secretion system and BopA are required for evasion of LC3-associated phagocytosisThe capsule is a virulence determinant in the pathogenesis of Pasteurella multocida M1404 (B:2).Acapsular Pasteurella multocida B:2 can stimulate protective immunity against pasteurellosis.Natural selection in the chicken host identifies 3-deoxy-D-manno-octulosonic acid kinase residues essential for phosphorylation of Pasteurella multocida lipopolysaccharide.A heptosyltransferase mutant of Pasteurella multocida produces a truncated lipopolysaccharide structure and is attenuated in virulence.Genomic scale analysis of Pasteurella multocida gene expression during growth within the natural chicken hostColistin resistance in Acinetobacter baumannii is mediated by complete loss of lipopolysaccharide production.Genomic evidence for a globally distributed, bimodal population in the ovine footrot pathogen Dichelobacter nodosus.Screening of 71 P. multocida proteins for protective efficacy in a fowl cholera infection model and characterization of the protective antigen PlpEDifferent surface charge of colistin-susceptible and -resistant Acinetobacter baumannii cells measured with zeta potential as a function of growth phase and colistin treatmentDevelopment of a rapid multiplex PCR assay to genotype Pasteurella multocida strains by use of the lipopolysaccharide outer core biosynthesis locus.Insertion sequence ISAba11 is involved in colistin resistance and loss of lipopolysaccharide in Acinetobacter baumannii.Novel approach to optimize synergistic carbapenem-aminoglycoside combinations against carbapenem-resistant Acinetobacter baumanniiStrategies for Intracellular Survival of Burkholderia pseudomallei.Burkholderia pseudomallei type III secretion system cluster 3 ATPase BsaS, a chemotherapeutic target for small-molecule ATPase inhibitors.Role for the Burkholderia pseudomallei type three secretion system cluster 1 bpscN gene in virulence.Necrotic enteritis-derived Clostridium perfringens strain with three closely related independently conjugative toxin and antibiotic resistance plasmids.The transcriptomic response of Acinetobacter baumannii to colistin and doripenem alone and in combination in an in vitro pharmacokinetics/pharmacodynamics model.Colistin-resistant, lipopolysaccharide-deficient Acinetobacter baumannii responds to lipopolysaccharide loss through increased expression of genes involved in the synthesis and transport of lipoproteins, phospholipids, and poly-β-1,6-N-acetylglucosaComparative Genomic Analysis of Asian Haemorrhagic Septicaemia-Associated Strains of Pasteurella multocida Identifies More than 90 Haemorrhagic Septicaemia-Specific Genes.The Burkholderia pseudomallei Proteins BapA and BapC Are Secreted TTSS3 Effectors and BapB Levels Modulate Expression of BopE.Perturbation of the two-component signal transduction system, BprRS, results in attenuated virulence and motility defects in Burkholderia pseudomallei.RNA-seq analysis of virR and revR mutants of Clostridium perfringens.Natural transformation of Gallibacterium anatisEffect of colistin exposure and growth phase on the surface properties of live Acinetobacter baumannii cells examined by atomic force microscopySynergistic killing of NDM-producing MDR Klebsiella pneumoniae by two 'old' antibiotics-polymyxin B and chloramphenicol.Decoration of Pasteurella multocida lipopolysaccharide with phosphocholine is important for virulenceHow does Pasteurella multocida respond to the host environment?Beclin 1 is required for starvation-enhanced, but not rapamycin-enhanced, LC3-associated phagocytosis of Burkholderia pseudomallei in RAW 264.7 cellsGlobal metabolic analyses identify key differences in metabolite levels between polymyxin-susceptible and polymyxin-resistant Acinetobacter baumannii.Lipopolysaccharide-deficient Acinetobacter baumannii shows altered signaling through host Toll-like receptors and increased susceptibility to the host antimicrobial peptide LL-37.Pasteurella multocida pathogenesis: 125 years after Pasteur.The fimbrial protein FlfA from Gallibacterium anatis is a virulence factor and vaccine candidate.Polymyxin Resistance in Acinetobacter baumannii: Genetic Mutations and Transcriptomic Changes in Response to Clinically Relevant Dosage Regimens.Identification of novel glycosyltransferases required for assembly of the Pasteurella multocida A:1 lipopolysaccharide and their involvement in virulence
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description
hulumtues
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onderzoeker
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researcher
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ricercatore
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հետազոտող
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name
John D Boyce
@nl
John D Boyce
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John D. Boyce
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John D. Boyce
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type
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John D Boyce
@nl
John D Boyce
@sl
John D. Boyce
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John D. Boyce
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prefLabel
John D Boyce
@nl
John D Boyce
@sl
John D. Boyce
@en
John D. Boyce
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no2018119267
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0000-0002-8614-3074