Cullin-3 mutation causes arterial stiffness and hypertension through a vascular smooth muscle mechanism.
about
Retinol-binding protein 7 is an endothelium-specific PPARγ cofactor mediating an antioxidant response through adiponectin.Mutant Cullin 3 causes familial hyperkalemic hypertension via dominant effects.Genetic Interference With Endothelial PPAR-γ (Peroxisome Proliferator-Activated Receptor-γ) Augments Effects of Angiotensin II While Impairing Responses to Angiotensin 1-7.PPARγ and retinol binding protein 7 form a regulatory hub promoting antioxidant properties of the endothelium.Nephron Remodeling Underlies Hyperkalemia in Familial Hyperkalemic Hypertension.Hypertension-Causing Mutation in Peroxisome Proliferator-Activated Receptor γ Impairs Nuclear Export of Nuclear Factor-κB p65 in Vascular Smooth Muscle.Mechanisms and controversies in mutant Cul3-mediated Familial Hyperkalemic Hypertension.Variants in the 3'-untranslated region of CUL3 is associated with risk of esophageal squamous cell carcinoma
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P2860
Cullin-3 mutation causes arterial stiffness and hypertension through a vascular smooth muscle mechanism.
description
2016 nî lūn-bûn
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2016年の論文
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2016年学术文章
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2016年学术文章
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2016年学术文章
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2016年学术文章
@zh-my
2016年学术文章
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2016年學術文章
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2016年學術文章
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2016年學術文章
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name
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
@en
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
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type
label
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
@en
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
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prefLabel
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
@en
Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
@nl
P2093
P2860
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Cullin-3 mutation causes arter ...... cular smooth muscle mechanism.
@en
P2093
Chunyan Hu
Deborah R Davis
Henry L Keen
Larry N Agbor
Stella-Rita C Ibeawuchi
P2860
P304
P356
10.1172/JCI.INSIGHT.91015
P577
2016-11-17T00:00:00Z