Loss of c-Met signaling sensitizes hepatocytes to lipotoxicity and induces cholestatic liver damage by aggravating oxidative stress.
about
Altered Met Receptor Phosphorylation and LRP1 Mediated Uptake in Cells Lacking Carbohydrate-Dependent Lysosomal Targeting.Genetic Nrf2 Overactivation Inhibits the Deleterious Effects Induced by Hepatocyte-Specific c-met Deletion during the Progression of NASH.Cholesterol overload in the liver aggravates oxidative stress-mediated DNA damage and accelerates hepatocarcinogenesis.
P2860
Loss of c-Met signaling sensitizes hepatocytes to lipotoxicity and induces cholestatic liver damage by aggravating oxidative stress.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 06 July 2016
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@en
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@nl
type
label
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@en
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@nl
prefLabel
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@en
Loss of c-Met signaling sensit ...... aggravating oxidative stress.
@nl
P2093
P2860
P50
P1433
P1476
Loss of c-Met signaling sensit ...... y aggravating oxidative stress
@en
P2093
Daekwan Seo
Elizabeth A Conner
Matthew Gillen
Mitsuteru Kitade
Seung-Bum Lee
Snorri S Thorgeirsson
Timo Gaiser
Valentina M Factor
Yun-Han Lee
P2860
P356
10.1016/J.TOX.2016.07.004
P407
P478
P577
2016-06-01T00:00:00Z