Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
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JARID1B is a luminal lineage-driving oncogene in breast cancerHistone demethylase lysine demethylase 5B in development and cancerBmi1 Is a Key Epigenetic Barrier to Direct Cardiac Reprogramming.KDM5B focuses H3K4 methylation near promoters and enhancers during embryonic stem cell self-renewal and differentiationH3K4 demethylase KDM5B regulates global dynamics of transcription elongation and alternative splicing in embryonic stem cellsHistone lysine demethylases in mammalian embryonic development.Epigenetic mechanisms of induced pluripotency.Impairment of preimplantation porcine embryo development by histone demethylase KDM5B knockdown through disturbance of bivalent H3K4me3-H3K27me3 modifications.The H3K4-Methyl Epigenome Regulates Leukemia Stem Cell Oncogenic Potential.Overexpression of JARID1B promotes differentiation via SHIP1/AKT signaling in human hypopharyngeal squamous cell carcinoma.The epigenomics of embryonic stem cell differentiation.SMYD5 regulates H4K20me3-marked heterochromatin to safeguard ES cell self-renewal and prevent spurious differentiation.JARID1B Enables Transit between Distinct States of the Stem-like Cell Population in Oral CancersRegulatory factors of induced pluripotency: current status.OCT4 supports extended LIF-independent self-renewal and maintenance of transcriptional and epigenetic networks in embryonic stem cells.Critical roles of protein methyltransferases and demethylases in the regulation of embryonic stem cell fate.Runt-Related Transcription Factor 2 Induction During Differentiation of Wharton's Jelly Mesenchymal Stem Cells to Osteoblasts Is Regulated by Jumonji AT-Rich Interactive Domain 1B Histone Demethylase.JARID1 Histone Demethylases: Emerging Targets in Cancer.SMYD5 Controls Heterochromatin and Chromosome Integrity during Embryonic Stem Cell Differentiation.Targeting JARID1B's demethylase activity blocks a subset of its functions in oral cancer.KDM5B decommissions the H3K4 methylation landscape of self-renewal genes during trophoblast stem cell differentiation.H4K20me3 co-localizes with activating histone modifications at transcriptionally dynamic regions in embryonic stem cells.Overexpression of KDM5B/JARID1B is associated with poor prognosis in hepatocellular carcinoma
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Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 07 October 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@en
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@nl
type
label
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@en
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@nl
prefLabel
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@en
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@nl
P2093
P2860
P356
P1476
Extended self-renewal and accelerated reprogramming in the absence of Kdm5b.
@en
P2093
Benjamin L Kidder
Chengyu Liu
P2860
P304
P356
10.1128/MCB.00692-13
P407
P50
P577
2013-10-07T00:00:00Z