Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
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Transformation from non-small-cell lung cancer to small-cell lung cancer: molecular drivers and cells of originKRAS-driven lung adenocarcinoma: combined DDR1/Notch inhibition as an effective therapyOvine pulmonary adenocarcinoma: a large animal model for human lung cancerTranscription factor Etv5 is essential for the maintenance of alveolar type II cells.Context-Dependent Effects of Amplified MAPK Signaling during Lung Adenocarcinoma Initiation and Progression.The cell of origin and subtype of K-Ras-induced lung tumors are modified by Notch and Sox2.Comparative pathobiology of environmentally induced lung cancers in humans and rodents.Inhibition of Phosphatidylcholine-Specific Phospholipase C Interferes with Proliferation and Survival of Tumor Initiating Cells in Squamous Cell CarcinomaAnalysis of lung tumor initiation and progression in transgenic mice for Cre-inducible overexpression of Cul4A gene.The cholesterol-binding protein NPC2 restrains recruitment of stromal macrophage-lineage cells to early-stage lung tumours.Engineering cancer stem-like cells from normal human lung epithelial cells.KLF4 regulates adult lung tumor-initiating cells and represses K-Ras-mediated lung cancer.One-step immortalization of primary human airway epithelial cells capable of oncogenic transformation.Diverse cells at the origin of lung adenocarcinoma.miR-494-3p is a novel tumor driver of lung carcinogenesis.Non-small-cell lung cancers: a heterogeneous set of diseases.Lineage factors and differentiation states in lung cancer progression.Overview of KRAS-Driven Genetically Engineered Mouse Models of Non-Small Cell Lung Cancer.Trp53 Mutants Drive Neuroendocrine Lung Cancer through Loss-of-Function Mechanisms with Gain-of-Function Effects on Chemotherapy Response.Modeling K-Ras-driven lung adenocarcinoma in mice: preclinical validation of therapeutic targets.Cell adhesion and polarity in squamous cell carcinoma of the lung.KRAS Allelic Imbalance Enhances Fitness and Modulates MAP Kinase Dependence in Cancer.Combined inhibition of DDR1 and Notch signaling is a therapeutic strategy for KRAS-driven lung adenocarcinoma.Molecular mechanisms of therapy resistance in solid tumors: chasing "moving" targets.Treatment with the nitric oxide synthase inhibitor L-NAME provides a survival advantage in a mouse model of Kras mutation-positive, non-small cell lung cancer.Development of Kras mutant lung adenocarcinoma in mice with knockout of the airway lineage-specific gene Gprc5a.Recombinant cells in the lung increase with age via de novo recombination events and clonal expansion.Lung Cancer Onset in Wild Type Mice Following Bone Marrow Reconstitution with kras(v12) Cells.Characterization of the cell of origin and propagation potential of the fibroblast growth factor 9-induced mouse model of lung adenocarcinoma.Molecular mechanisms of pathological tumor transformation and their clinical implications: predictors of pulmonary adenocarcinoma transformation into small cell carcinoma.Combining Cell Type-Restricted Adenoviral Targeting with Immunostaining and Flow Cytometry to Identify Cells-of-Origin of Lung Cancer.A Braf kinase-inactive mutant induces lung adenocarcinoma.Role and regulation of Yap in KrasG12D-induced lung cancer.[Advances in the Transformation to Small Cell Lung Cancer from Non-small Cell Lung Cancer Following Acquired Drug-resistance to EGFR Tyrosine Kinase Inhibitors].Inflammatory memory sensitizes skin epithelial stem cells to tissue damage.K-Ras(V14I) -induced Noonan syndrome predisposes to tumour development in mice.Application of the Next-Generation Sequencing Technology to Reveal Mechanism of Small Cell Lung Cancer Transformation from Adenocarcinoma.[Clinical Observation of Translating to Small Cell Lung Cancer Following Treatment with EGFR-Tyrosine Kinase Inhibitors in Lung Adenocarcinoma].Mitotic polarization of transcription factors during asymmetric division establishes fate of forming cancer cells.Lower expression level of IL-33 is associated with poor prognosis of pulmonary adenocarcinoma.
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Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 23 December 2013
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@en
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@nl
type
label
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@en
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@nl
prefLabel
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@en
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma.
@nl
P2093
P2860
P50
P356
P1476
Identification of cancer initiating cells in K-Ras driven lung adenocarcinoma
@en
P2093
Mariano Barbacid
Nieves Mijimolle
Sara Mainardi
P2860
P304
P356
10.1073/PNAS.1320383110
P407
P577
2013-12-23T00:00:00Z