Over-expression of Slit2 induces vessel formation and changes blood vessel permeability in mouse brain. - Test2 - elhamkhani - TriplyDB

Over-expression of Slit2 induces vessel formation and changes blood vessel permeability in mouse brain.

Over-expression of Slit2 induces vessel formation and changes blood vessel permeability in mouse brain.

http://www.wikidata.org/entity/Q37476754

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Effects of different tissue microenvironments on gene expression in breast cancer cells Slit2 overexpression results in increased microvessel density and lesion size in mice with induced endometriosis.Rpb3 promotes hepatocellular carcinoma through its N-terminus.Robo1 modulates proliferation and neurogenesis in the developing neocortex.Alpha-2 macroglobulin in Alzheimer's disease: a marker of neuronal injury through the RCAN1 pathway.The role of Slit-Robo signaling in the regulation of tissue barriers.Overexpression of Slit2 improves function of the paravascular pathway in the aging mouse brain

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Over-expression of Slit2 induces vessel formation and changes blood vessel permeability in mouse brain.

http://www.wikidata.org/entity/Q37476754

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 10 October 2011

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

@da

vědecký článek

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name

Over-expression of Slit2 induc ...... l permeability in mouse brain.

@en

Over-expression of Slit2 induc ...... l permeability in mouse brain.

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type

label

Over-expression of Slit2 induc ...... l permeability in mouse brain.

@en

Over-expression of Slit2 induc ...... l permeability in mouse brain.

@nl

prefLabel

Over-expression of Slit2 induc ...... l permeability in mouse brain.

@en

Over-expression of Slit2 induc ...... l permeability in mouse brain.

@nl

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Acta Pharmacologica Sinica

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Over-expression of Slit2 induc ...... l permeability in mouse brain.

@en

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Hai-xiong Han

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Jian-guo Geng

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Slit proteins bind Robo receptors and have an evolutionarily conserved role in repulsive axon guidance

The neuronal repellent Slit inhibits leukocyte chemotaxis induced by chemotactic factors

Neuronal chemorepellent Slit2 inhibits vascular smooth muscle cell migration by suppressing small GTPase Rac1 activation

HDAC5 is a repressor of angiogenesis and determines the angiogenic gene expression pattern of endothelial cells

Repulsive axon guidance molecule Slit3 is a novel angiogenic factor

The axonal repellent, Slit2, inhibits directional migration of circulating neutrophils

Roundabout controls axon crossing of the CNS midline and defines a novel subfamily of evolutionarily conserved guidance receptors

Vertebrate slit, a secreted ligand for the transmembrane protein roundabout, is a repellent for olfactory bulb axons

Repulsive axon guidance: Abelson and Enabled play opposing roles downstream of the roundabout receptor

Induction of tumor angiogenesis by Slit-Robo signaling and inhibition of cancer growth by blocking Robo activity

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1327-1336

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scholarly article

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10.1038/APS.2011.106

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11

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32

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2011-10-10T00:00:00Z

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51706192

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1048321741

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21986575

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3891311

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