IL-23 is critical for induction of arthritis, osteoclast formation, and maintenance of bone mass.
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Animal Models of Bone Loss in Inflammatory Arthritis: from Cytokines in the Bench to Novel Treatments for Bone Loss in the Bedside-a Comprehensive ReviewOsteoimmunology and the influence of pro-inflammatory cytokines on osteoclastsReview: The interleukin-23/interleukin-17 axis in spondyloarthritis pathogenesis: Th17 and beyondInteractions of the Immune System with Skin and Bone Tissue in Psoriatic Arthritis: A Comprehensive Review.A new strategy to deliver synthetic protein drugs: self-reproducible biologics using minicircles.Cytokine-mediated bone destruction in rheumatoid arthritis.A novel in vivo gene transfer technique and in vitro cell based assays for the study of bone loss in musculoskeletal disordersIL-17A gene transfer induces bone loss and epidermal hyperplasia associated with psoriatic arthritis.Leukotriene B4 activates intracellular calcium and augments human osteoclastogenesis.A Dual Target-directed Agent against Interleukin-6 Receptor and Tumor Necrosis Factor α ameliorates experimental arthritis.Rheumatoid and pyrophosphate arthritis synovial fibroblasts induce osteoclastogenesis independently of RANKL, TNF and IL-6.Bone effects of biologic drugs in rheumatoid arthritis.Tristetraprolin regulation of interleukin 23 mRNA stability prevents a spontaneous inflammatory diseaseThe IL-23/IL-17 axis in psoriatic arthritis.Advances in osteoimmunology: pathophysiologic concepts and treatment opportunities.Th17 Cells in Immunopathogenesis and treatment of rheumatoid arthritis.Mechanistic insights from animal models of psoriasis and psoriatic arthritis.Alternative pathways of osteoclastogenesis in inflammatory arthritis.The Role of T Helper 17 Cells and Other IL-17-Producing Cells in Bone Resorption and Remodeling.The IL-23-IL-17 axis in inflammatory arthritis.Interleukin-23 is sufficient to induce rapid de novo gut tumorigenesis, independent of carcinogens, through activation of innate lymphoid cells.Review: animal models as a tool to dissect pivotal pathways driving spondyloarthritis.Non-Canonical (RANKL-Independent) Pathways of Osteoclast Differentiation and Their Role in Musculoskeletal Diseases.Calcium channels: the potential therapeutic targets for inflammatory bone destruction of rheumatoid arthritis.New insights toward the pathogenesis of ankylosing spondylitis; genetic variations and epigenetic modifications.Critical Role of LTB4/BLT1 in IL-23-Induced Synovial Inflammation and Osteoclastogenesis via NF-κB.Concise Review: Stem Cells in Osteoimmunology.T Cell-Independent Mechanisms Associated with Neutrophil Extracellular Trap Formation and Selective Autophagy in IL-17A-Mediated Epidermal Hyperplasia.Autoreactive Th1 cells activate monocytes to support regional Th17 responses in inflammatory arthritis.Continuous IL-23 stimulation drives ILC3 depletion in the upper GI tract and, in combination with TNFα, induces robust activation and a phenotypic switch of ILC3.Crosstalk among IL-23 and DNAX activating protein of 12 kDa-dependent pathways promotes osteoclastogenesisAutoimmune or Autoiflammatory? Bad to the Bone.In situ analysis of interleukin-23- and interleukin-12-positive cells in the spine of patients with ankylosing spondylitis.Enthesitis: from pathophysiology to treatment.A novel ex vivo culture model for inflammatory bone destruction.The role of IL-23 receptor signaling in inflammation-mediated erosive autoimmune arthritis and bone remodeling.Progress of genome-wide association studies of ankylosing spondylitis.Interleukin-23-Dependent γ/δ T Cells Produce Interleukin-17 and Accumulate in the Enthesis, Aortic Valve, and Ciliary Body in Mice.Axial spondyloarthritis.γδTCR regulates production of interleukin-27 by neutrophils and attenuates inflammatory arthritis.
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IL-23 is critical for induction of arthritis, osteoclast formation, and maintenance of bone mass.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
@pt
bilimsel makale
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scientific article published on 13 June 2011
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
IL-23 is critical for inductio ...... and maintenance of bone mass.
@en
IL-23 is critical for inductio ...... and maintenance of bone mass.
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type
label
IL-23 is critical for inductio ...... and maintenance of bone mass.
@en
IL-23 is critical for inductio ...... and maintenance of bone mass.
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prefLabel
IL-23 is critical for inductio ...... and maintenance of bone mass.
@en
IL-23 is critical for inductio ...... and maintenance of bone mass.
@nl
P2093
P2860
P356
P1476
IL-23 is critical for inductio ...... , and maintenance of bone mass
@en
P2093
Cheng-Chi Chao
Chuan-Chu Chou
Dan Gorman
Drake Laface
Edward P Bowman
Marlowe Tessmer
Mary Petro
Nancy E Lane
Robert H Pierce
Sarvesh Adda
P2860
P304
P356
10.4049/JIMMUNOL.1003986
P407
P577
2011-06-13T00:00:00Z