Administration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancy.
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Preeclampsia: long-term consequences for vascular healthInflammation, immunity, and hypertensive end-organ damageInflammation and hypertension: new understandings and potential therapeutic targetsNeutrophil Depletion Attenuates Placental Ischemia-Induced Hypertension in the RatA Dormant Microbial Component in the Development of PreeclampsiaBioactive factors in uteroplacental and systemic circulation link placental ischemia to generalized vascular dysfunction in hypertensive pregnancy and preeclampsiaDual opposing roles of adaptive immunity in hypertension.The role of inflammation in the pathology of preeclampsiaSex differences in T cells in hypertension.Autoimmunity: an underlying factor in the pathogenesis of hypertension.Role of IL-17 Variants in Preeclampsia in Chinese Han Women.Placental ischemia increases seizure susceptibility and cerebrospinal fluid cytokinesIL-10 supplementation increases Tregs and decreases hypertension in the RUPP rat model of preeclampsia.Mechanisms of Endothelial Dysfunction in Hypertensive Pregnancy and Preeclampsia.Identifying immune mechanisms mediating the hypertension during preeclampsiaTH17- and IL-17- mediated autoantibodies and placental oxidative stress play a role in the pathophysiology of pre-eclampsiaPlacental Ischemia and Resultant Phenotype in Animal Models of Preeclampsia.Magnesium Sulfate Prevents Placental Ischemia-Induced Increases in Brain Water Content and Cerebrospinal Fluid Cytokines in Pregnant Rats.Oxidative Stress: Placenta Function and Dysfunction.Proliferation of endogenous regulatory T cells improve the pathophysiology associated with placental ischaemia of pregnancy.Oxidative Stress and Hypertensive Diseases.IL-17 mediates neutrophil infiltration and renal fibrosis following recovery from ischemia reperfusion: compensatory role of natural killer cells in athymic rats.Hypertension in an Animal Model of HELLP Syndrome is Associated With Activation of Endothelin 1.Circulating Th1, Th2, and Th17 Levels in Hypertensive Patients.Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancy.Matrix Metalloproteinases in Normal Pregnancy and Preeclampsia.Th17 cells contribute to pulmonary fibrosis and inflammation during chronic kidney disease progression following acute ischemia.Role of Immune Factors in Angiotensin II-Induced Hypertension and Renal Damage in Dahl Salt-Sensitive Rats.Preeclampsia: From Inflammation to Immunoregulation.
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P2860
Administration of interleukin-17 soluble receptor C suppresses TH17 cells, oxidative stress, and hypertension in response to placental ischemia during pregnancy.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on 23 September 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
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vědecký článek
@cs
name
Administration of interleukin- ...... tal ischemia during pregnancy.
@en
Administration of interleukin- ...... tal ischemia during pregnancy.
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type
label
Administration of interleukin- ...... tal ischemia during pregnancy.
@en
Administration of interleukin- ...... tal ischemia during pregnancy.
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prefLabel
Administration of interleukin- ...... tal ischemia during pregnancy.
@en
Administration of interleukin- ...... tal ischemia during pregnancy.
@nl
P2093
P2860
P50
P1433
P1476
Administration of interleukin- ...... tal ischemia during pregnancy.
@en
P2093
Babbette LaMarca
Denise C Cornelius
James P Hogg
Janae Moseley
Jeremy Scott
P2860
P304
P356
10.1161/HYPERTENSIONAHA.113.01514
P407
P577
2013-09-23T00:00:00Z