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Communications between Mitochondria, the Nucleus, Vacuoles, Peroxisomes, the Endoplasmic Reticulum, the Plasma Membrane, Lipid Droplets, and the Cytosol during Yeast Chronological AgingAutophagy regulates sphingolipid levels in the liverDrug synergy drives conserved pathways to increase fission yeast lifespanSix plant extracts delay yeast chronological aging through different signaling pathwaysThe effects of graded levels of calorie restriction: IX. Global metabolomic screen reveals modulation of carnitines, sphingolipids and bile acids in the liver of C57BL/6 mice.Mechanisms Underlying the Essential Role of Mitochondrial Membrane Lipids in Yeast Chronological AgingSpermidine feeding decreases age-related locomotor activity loss and induces changes in lipid composition.Mechanisms underlying the anti-aging and anti-tumor effects of lithocholic bile acidNutrient sensing and signaling in the yeast Saccharomyces cerevisiae.Harnessing the power of yeast to elucidate the role of sphingolipids in metabolic and signaling processes pertinent to psychiatric disorders.Producing human ceramide-NS by metabolic engineering using yeast Saccharomyces cerevisiae.Demographic and clinical variables affecting mid- to late-life trajectories of plasma ceramide and dihydroceramide speciesSphingolipids and mitochondrial function in budding yeast.Resveratrol and its oligomers: modulation of sphingolipid metabolism and signaling in disease.Autophagic flux and autophagosome morphogenesis require the participation of sphingolipids.Exploring the power of yeast to model aging and age-related neurodegenerative disorders.The Association Between Plasma Ceramides and Sphingomyelins and Risk of Alzheimer's Disease Differs by Sex and APOE in the Baltimore Longitudinal Study of Aging.Stress-Activated Degradation of Sphingolipids Regulates Mitochondrial Function and Cell Death in Yeast.Benzoxazolone carboxamides: potent and systemically active inhibitors of intracellular acid ceramidase.Loss of Sphingosine Kinase Alters Life History Traits and Locomotor Function in Caenorhabditis elegans.Caloric restriction extends yeast chronological lifespan via a mechanism linking cellular aging to cell cycle regulation, maintenance of a quiescent state, entry into a non-quiescent state and survival in the non-quiescent state.Metabolic remodeling in human colorectal cancer and surrounding tissues: alterations in regulation of mitochondrial respiration and metabolic fluxes.Sphingolipids and their metabolism in physiology and disease.Some Metabolites Act as Second Messengers in Yeast Chronological Aging.Mechanisms through which lithocholic acid delays yeast chronological aging under caloric restriction conditions
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on 15 August 2013
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Sphingolipids and lifespan regulation.
@en
Sphingolipids and lifespan regulation.
@nl
type
label
Sphingolipids and lifespan regulation.
@en
Sphingolipids and lifespan regulation.
@nl
prefLabel
Sphingolipids and lifespan regulation.
@en
Sphingolipids and lifespan regulation.
@nl
P2093
P2860
P1476
Sphingolipids and lifespan regulation.
@en
P2093
Bradley R Withers
Robert C Dickson
Xinhe Huang
P2860
P304
P356
10.1016/J.BBALIP.2013.08.006
P407
P577
2013-08-15T00:00:00Z