MET exon 14 juxtamembrane splicing mutations: clinical and therapeutical perspectives for cancer therapy. - Test2 - elhamkhani - TriplyDB

MET exon 14 juxtamembrane splicing mutations: clinical and therapeutical perspectives for cancer therapy.

MET exon 14 juxtamembrane splicing mutations: clinical and therapeutical perspectives for cancer therapy.

http://www.wikidata.org/entity/Q37597858

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Hepatocyte Growth Factor/c-Met Signaling in Head and Neck Cancer and Implications for Treatment Activated HGF-c-Met Axis in Head and Neck Cancer.Molecular Diagnostics in Clinical Oncology

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P2860

MET exon 14 juxtamembrane splicing mutations: clinical and therapeutical perspectives for cancer therapy.

http://www.wikidata.org/entity/Q37597858

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on January 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

@en

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

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type

label

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

@en

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

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prefLabel

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

@en

MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

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Annals of translational medicine

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MET exon 14 juxtamembrane spli ...... rspectives for cancer therapy.

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Giampaolo Tortora

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An overview of the c-MET signaling pathway

Comprehensive genomic characterization defines human glioblastoma genes and core pathways

Understanding and Targeting MET Signaling in Solid Tumors - Are We There Yet?

Met in urological cancers

Durable complete response of metastatic gastric cancer with anti-Met therapy followed by resistance at recurrence.

Phase II and biomarker study of the dual MET/VEGFR2 inhibitor foretinib in patients with papillary renal cell carcinoma

Activation of MET via diverse exon 14 splicing alterations occurs in multiple tumor types and confers clinical sensitivity to MET inhibitors.

Response to MET inhibitors in patients with stage IV lung adenocarcinomas harboring MET mutations causing exon 14 skipping

MET Exon 14 Mutations in Non-Small-Cell Lung Cancer Are Associated With Advanced Age and Stage-Dependent MET Genomic Amplification and c-Met Overexpression.

The clinical and functional significance of c-Met in breast cancer: a review

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2

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scholarly article

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10.21037/ATM.2016.12.33

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1

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Luisa Carbognin

Anastasios Gkountakos

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2017-01-01T00:00:00Z

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28164087

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5253296

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