Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis. - Test2 - elhamkhani - TriplyDB

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

http://www.wikidata.org/entity/Q37617215

about

Gut Microbiota Regulates K/BxN Autoimmune Arthritis through Follicular Helper T but Not Th17 Cells.PTPN22 Is a Critical Regulator of Fcγ Receptor-Mediated Neutrophil Activation Variable Colonization after Reciprocal Fecal Microbiota Transfer between Mice with Low and High Richness Microbiota.Low-dose penicillin exposure in early life decreases Th17 and the susceptibility to DSS colitis in mice through gut microbiota modification.Interleukin 17-expressing Innate Synovial Cells Drive K/Bxn Serum-induced Arthritis.Gut Microbiota and IL-17A: Physiological and Pathological Responses.Intestinal barrier and gut microbiota: Shaping our immune responses throughout life.

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Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

http://www.wikidata.org/entity/Q37617215

description

article científic

@ca

article scientifique

@fr

articolo scientifico

@it

artigo científico

@pt

bilimsel makale

@tr

scientific article published on 21 February 2014

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@en

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@nl

type

label

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@en

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@nl

prefLabel

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@en

Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@nl

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IN.2014.14.1.38

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Gut-residing Microbes Alter the Host Susceptibility to Autoantibody-mediated Arthritis.

@en

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A Reum Lee

http://www.w3.org/2001/XMLSchema#string

Bo-Eun Jin

http://www.w3.org/2001/XMLSchema#string

Dong Soo Han

http://www.w3.org/2001/XMLSchema#string

Eunkyeong Jang

http://www.w3.org/2001/XMLSchema#string

Ho-Youn Kim

http://www.w3.org/2001/XMLSchema#string

Hyerim Lee

http://www.w3.org/2001/XMLSchema#string

Jeehee Youn

http://www.w3.org/2001/XMLSchema#string

P2860

Specific microbiota direct the differentiation of IL-17-producing T-helper cells in the mucosa of the small intestine

Interactions between the microbiota and the immune system

IL-17 and Th17 Cells

Induction of intestinal Th17 cells by segmented filamentous bacteria

A microbial symbiosis factor prevents intestinal inflammatory disease

Gut-residing segmented filamentous bacteria drive autoimmune arthritis via T helper 17 cells

Interactions between commensal intestinal bacteria and the immune system

From systemic T cell self-reactivity to organ-specific autoimmune disease via immunoglobulins.

Proinflammatory T helper type 17 cells are effective B-cell helpers

Th17 cells can provide B cell help in autoantibody induced arthritis.

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38-44

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scholarly article

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10.4110/IN.2014.14.1.38

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1

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2014-02-21T00:00:00Z

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260612273

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24605079

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