Specific contactin N-glycans are implicated in neurofascin binding and autoimmune targeting in peripheral neuropathies.
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Neuroinflammation in the peripheral nerve: Cause, modulator, or bystander in peripheral neuropathies?Mapping autoantigen epitopes: molecular insights into autoantibody-associated disorders of the nervous systemChronic inflammatory demyelinating polyradiculoneuropathy: from pathology to phenotype.Subtype-specific therapy for autoimmune neuropathies?Contactin 1 IgG4 associates to chronic inflammatory demyelinating polyneuropathy with sensory ataxiaNeurofascin-155 IgG4 in chronic inflammatory demyelinating polyneuropathy.Dopamine-2 receptor extracellular N-terminus regulates receptor surface availability and is the target of human pathogenic antibodies from children with movement and psychiatric disorders.Optimizing IgG therapy in chronic autoimmune neuropathies: a hypothesis driven approach.Autoimmune antigenic targets at the node of Ranvier in demyelinating disorders.Progress in inflammatory neuropathy -the legacy of Dr Jack Griffin.Autoantibodies in chronic inflammatory neuropathies: diagnostic and therapeutic implications.Inhibitory axons are targeted in hippocampal cell culture by anti-Caspr2 autoantibodies associated with limbic encephalitis.Cellular investigations with human antibodies associated with the anti-IgLON5 syndrome.Rituximab in treatment-resistant CIDP with antibodies against paranodal proteins.Antibodies against peripheral nerve antigens in chronic inflammatory demyelinating polyradiculoneuropathy.Neurofascin antibodies in autoimmune, genetic, and idiopathic neuropathies.ARTHUR ASBURY LECTURE: Chronic inflammatory demyelinating polyradiculoneuropathy: clinical aspects and new animal models of auto-immunity to nodal components.IgG4-mediated autoimmune diseases: a niche of antibody-mediated disorders.Auto-antibodies to contactin-associated protein 1 (Caspr) in two patients with painful inflammatory neuropathy.Mechanisms of Caspr2 antibodies in autoimmune encephalitis and neuromyotonia.Destruction of paranodal architecture in inflammatory neuropathy with anti-contactin-1 autoantibodies.A New Classification System for IgG4 Autoantibodies.Contactin-1 IgG4 antibodies cause paranode dismantling and conduction defects.Autoantibodies Against the Node of Ranvier in Seropositive Chronic Inflammatory Demyelinating Polyneuropathy: Diagnostic, Pathogenic, and Therapeutic Relevance.
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P2860
Specific contactin N-glycans are implicated in neurofascin binding and autoimmune targeting in peripheral neuropathies.
description
article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 04 February 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
@en
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
@nl
type
label
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
@en
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
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prefLabel
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
@en
Specific contactin N-glycans a ...... ng in peripheral neuropathies.
@nl
P2093
P2860
P50
P356
P1476
Specific contactin N-glycans a ...... ing in peripheral neuropathies
@en
P2093
Bruno Hivert
Isabel Illa
Luis Querol
P2860
P304
P356
10.1074/JBC.M113.528489
P407
P577
2014-02-04T00:00:00Z