Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells. - Test2 - elhamkhani - TriplyDB

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

http://www.wikidata.org/entity/Q37645001

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Cellular glycosylation senses metabolic changes and modulates cell plasticity during epithelial to mesenchymal transition.The significance of glypican-3 expression profiling in the tumor cellular origin theoretical system for hepatocellular carcinoma progression.

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Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

http://www.wikidata.org/entity/Q37645001

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

@pt

bilimsel makale

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scientific article published on 06 August 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

@en

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

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type

label

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

@en

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

@nl

prefLabel

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

@en

Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells.

@nl

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ONCOTARGET.11107

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Glypican-3 induces a mesenchymal to epithelial transition in human breast cancer cells

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Ana María Cabanillas

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Ancely Ferreira Dos Santos

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Elisa Bal de Kier Joffé

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Gisela Novack

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Leticia Labriola

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Lilian Fedra Castillo

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María Amparo Lago Huvelle

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María Candelaria Llorens

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Rocío Tascón

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A molecular mechanism for the effect of lithium on development

Implications of the Hybrid Epithelial/Mesenchymal Phenotype in Metastasis

Glypican-3: a marker and a therapeutic target in hepatocellular carcinoma

Molecular mechanisms of epithelial-mesenchymal transition

Glypicans in growth control and cancer

Reduction of beta-catenin/T-cell transcription factor signaling by aspirin and indomethacin is caused by an increased stabilization of phosphorylated beta-catenin

Expression of the cell surface proteoglycan glypican-5 is developmentally regulated in kidney, limb, and brain

The loss of glypican-3 induces alterations in Wnt signaling

T3-activation of the rat growth hormone gene is inhibited by a zinc finger/homeodomain protein

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60133-60154

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scholarly article

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10.18632/ONCOTARGET.11107

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37

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7

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Jorge Eduardo Shortrede

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2016-09-01T00:00:00Z

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27507057

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5312374

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