Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
about
Identification of human proteins that modify misfolding and proteotoxicity of pathogenic ataxin-1Using C. elegans to discover therapeutic compounds for ageing-associated neurodegenerative diseasesRegulation of protein homeostasis in neurodegenerative diseases: the role of coding and non-coding genesMesenchymal stem cells-based therapy as a potential treatment in neurodegenerative disorders: is the escape from senescence an answer?Neuronal reprograming of protein homeostasis by calcium-dependent regulation of the heat shock responseA genetic screening strategy identifies novel regulators of the proteostasis networkThe SUN protein Mps3 is required for spindle pole body insertion into the nuclear membrane and nuclear envelope homeostasisMammalian genes induce partially reprogrammed pluripotent stem cells in non-mammalian vertebrate and invertebrate speciesCaenorhabditis elegans dnj-14, the orthologue of the DNAJC5 gene mutated in adult onset neuronal ceroid lipofuscinosis, provides a new platform for neuroprotective drug screening and identifies a SIR-2.1-independent action of resveratrol.Ethosuximide ameliorates neurodegenerative disease phenotypes by modulating DAF-16/FOXO target gene expressionRAS-MAPK-MSK1 pathway modulates ataxin 1 protein levels and toxicity in SCA1Modifiers of mutant huntingtin aggregation: functional conservation of C. elegans-modifiers of polyglutamine aggregation.Identification of common genetic modifiers of neurodegenerative diseases from an integrative analysis of diverse genetic screens in model organismsIdentification of mammalian protein quality control factors by high-throughput cellular imagingA yeast model for polyalanine-expansion aggregation and toxicityThe Copper Metabolism MURR1 domain protein 1 (COMMD1) modulates the aggregation of misfolded protein species in a client-specific mannerA new role for laminins as modulators of protein toxicity in Caenorhabditis elegans.Morphological remodeling of C. elegans neurons during aging is modified by compromised protein homeostasis.Genetic modifiers of neurological diseaseRNA-binding proteins with prion-like domains in ALS and FTLD-U.Polyglutamine misfolding in yeast: toxic and protective aggregation.Brain activation of SIRT1: role in neuropathology.Experimental models for identifying modifiers of polyglutamine-induced aggregation and neurodegeneration.Optofluidic holographic microscopy with custom field of view (FoV) using a linear array detector.Transforming Growth Factor-Beta Signaling in the Neural Stem Cell Niche: A Therapeutic Target for Huntington's Disease.Detection of amyloid fibrils in Parkinson's disease using plasmonic chirality.ANKRD16 prevents neuron loss caused by an editing-defective tRNA synthetase
P2860
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P2860
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
description
2009 nî lūn-bûn
@nan
2009年の論文
@ja
2009年学术文章
@wuu
2009年学术文章
@zh-cn
2009年学术文章
@zh-hans
2009年学术文章
@zh-my
2009年学术文章
@zh-sg
2009年學術文章
@yue
2009年學術文章
@zh
2009年學術文章
@zh-hant
name
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@en
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@nl
type
label
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@en
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@nl
altLabel
Neurodegenerative diseases: Lessons from genome‐wide screens in small model organisms
@en
prefLabel
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@en
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@nl
P2860
P50
P356
P1476
Neurodegenerative diseases: Lessons from genome-wide screens in small model organisms.
@en
P2093
Ellen A A Nollen
P2860
P304
P356
10.1002/EMMM.200900051
P577
2009-11-01T00:00:00Z