Rapid degradation of mutant SLC25A46 by the ubiquitin-proteasome system results in MFN1/2-mediated hyperfusion of mitochondria
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Mitochondrial Membrane Dynamics and Inherited Optic Neuropathies.Loss of function of SLC25A46 causes lethal congenital pontocerebellar hypoplasia.Loss of SLC25A46 causes neurodegeneration by affecting mitochondrial dynamics and energy production in mice.Regulation of Mitochondrial Dynamics by Proteolytic Processing and Protein Turnover.UBXD1 is a mitochondrial recruitment factor for p97/VCP and promotes mitophagy
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Rapid degradation of mutant SLC25A46 by the ubiquitin-proteasome system results in MFN1/2-mediated hyperfusion of mitochondria
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article científic
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article scientifique
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articolo scientifico
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artigo científico
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bilimsel makale
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scientific article published on 05 January 2017
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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Rapid degradation of mutant SL ...... ed hyperfusion of mitochondria
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Rapid degradation of mutant SL ...... d hyperfusion of mitochondria.
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type
label
Rapid degradation of mutant SL ...... ed hyperfusion of mitochondria
@en
Rapid degradation of mutant SL ...... d hyperfusion of mitochondria.
@nl
prefLabel
Rapid degradation of mutant SL ...... ed hyperfusion of mitochondria
@en
Rapid degradation of mutant SL ...... d hyperfusion of mitochondria.
@nl
P2093
P2860
P356
P1476
Rapid degradation of mutant SL ...... ed hyperfusion of mitochondria
@en
P2093
Ajay A Vashisht
James A Wohlschlegel
Janos Steffen
Joanna C Jen
P2860
P304
P356
10.1091/MBC.E16-07-0545
P577
2017-01-05T00:00:00Z