Functionally distinct PI 3-kinase pathways regulate myelination in the peripheral nervous system.
about
Molecular control of the neural crest and peripheral nervous system developmentNew insights on Schwann cell developmentLaminin 211 inhibits protein kinase A in Schwann cells to modulate neuregulin 1 type III-driven myelinationmTORC1 is essential for early steps during Schwann cell differentiation of amniotic fluid stem cells and regulates lipogenic gene expressionRole of ERK1/2 MAPK signaling in the maintenance of myelin and axonal integrity in the adult CNS.Dynamic regulation of Schwann cell enhancers after peripheral nerve injuryRegulation of Peripheral Nerve Myelin Maintenance by Gene Repression through Polycomb Repressive Complex 2Akt Regulates Axon Wrapping and Myelin Sheath Thickness in the PNSRab27a/Slp2-a complex is involved in Schwann cell myelination.AlphaB-crystallin regulates remyelination after peripheral nerve injury.Neuregulin-ERBB signaling in the nervous system and neuropsychiatric diseasesMolecular signaling mechanisms of axon-glia communication in the peripheral nervous system.Schwann cell myelinationIn vitro and in vivo inhibition of mTOR by 1,25-dihydroxyvitamin D3 to improve early diabetic nephropathy via the DDIT4/TSC2/mTOR pathway.Dual function of the PI3K-Akt-mTORC1 axis in myelination of the peripheral nervous system.Lithium Reversibly Inhibits Schwann Cell Proliferation and Differentiation Without Inducing Myelin Loss.Effects of Paclitaxel and Eribulin in Mouse Sciatic Nerve: A Microtubule-Based Rationale for the Differential Induction of Chemotherapy-Induced Peripheral Neuropathy.Implications of Schwann Cells Biomechanics and Mechanosensitivity for Peripheral Nervous System Physiology and Pathophysiology.Deficiency of a membrane skeletal protein, 4.1G, results in myelin abnormalities in the peripheral nervous system.Myelination and mTOR.Myelinating Schwann Cell Polarity and Mechanically-Driven Myelin Sheath Elongation.Multiple lines of inhibitory feedback on AKT kinase in Schwann cells lacking TSC1/2 hint at distinct functions of mTORC1 and AKT in nerve development.mTORC1 is transiently reactivated in injured nerves to promote c-Jun elevation and Schwann cell dedifferentiation.Pathologic Remodeling of Endoneurial Tubules in Human Neuromas.
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P2860
Functionally distinct PI 3-kinase pathways regulate myelination in the peripheral nervous system.
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on March 2014
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@en
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@nl
type
label
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@en
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@nl
prefLabel
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@en
Functionally distinct PI 3-kin ...... the peripheral nervous system.
@nl
P2093
P2860
P356
P1476
Functionally distinct PI 3-kin ...... the peripheral nervous system
@en
P2093
Aniko Naray Fejes-Toth
Bradley A Heller
David Chandler
Ethan Grund
Filippo Giancotti
Florian Lang
Gerard Fejes-Toth
Grant Morahan
Jakob Voelkl
Luba Kalaydjieva
P2860
P304
P356
10.1083/JCB.201307057
P407
P577
2014-03-01T00:00:00Z