Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium. - Test2 - elhamkhani - TriplyDB

Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium.

Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium.

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Phenotyping of Mice with Heart Specific Overexpression of A2A-Adenosine Receptors: Evidence for Cardioprotective Effects of A2A-Adenosine Receptors.

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Transcriptomic effects of adenosine 2A receptor deletion in healthy and endotoxemic murine myocardium.

http://www.wikidata.org/entity/Q37677794

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 30 September 2016

@en

vedecký článok

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vetenskaplig artikel

@sv

videnskabelig artikel

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vědecký článek

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name

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

@en

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

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type

label

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

@en

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

@nl

prefLabel

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

@en

Transcriptomic effects of aden ...... endotoxemic murine myocardium.

@nl

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Purinergic Signalling

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Transcriptomic effects of aden ...... endotoxemic murine myocardium.

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Bunyen Teng

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Catherine Ledent

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John P Headrick

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Lea M D Delbridge

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Melissa E Reichelt

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Polly A Hofmann

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R Ray Morrison

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S Jamal Mustafa

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Aggressiveness, hypoalgesia and high blood pressure in mice lacking the adenosine A2a receptor

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Exploration, normalization, and summaries of high density oligonucleotide array probe level data

Stimulation of signal transducer and activator of transcription-1 (STAT1)-dependent gene transcription by lipopolysaccharide and interferon-gamma is regulated by mammalian target of rapamycin

S100A8 and S100A9 mediate endotoxin-induced cardiomyocyte dysfunction via the receptor for advanced glycation end products

MCP-1-induced protein attenuates endotoxin-induced myocardial dysfunction by suppressing cardiac NF-кB activation via inhibition of IкB kinase activation

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scholarly article

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