Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord. - Test2 - elhamkhani - TriplyDB

Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord.

Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord.

http://www.wikidata.org/entity/Q37702402

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Neurochemical characterization of pERK-expressing spinal neurons in histamine-induced itch.Spinal neurons that contain gastrin-releasing peptide seldom express Fos or phosphorylate extracellular signal-regulated kinases in response to intradermal chloroquine.Role of ERK1/2 activation on itch sensation induced by bradykinin B1 activation in inflamed skin.Preprotachykinin A is expressed by a distinct population of excitatory neurons in the mouse superficial spinal dorsal horn including cells that respond to noxious and pruritic stimuli.Antioxidants Attenuate Acute and Chronic Itch: Peripheral and Central Mechanisms of Oxidative Stress in Pruritus.Spinal SET7/9 may contribute to the maintenance of cancer-induced bone pain in mice.The behavioral study on the interactive aggravation between pruritus and depression.NMDA receptor antagonists attenuate intrathecal morphine-induced pruritus through ERK phosphorylation.

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Extracellular signal-regulated kinase (ERK) activation is required for itch sensation in the spinal cord.

http://www.wikidata.org/entity/Q37702402

description

article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 03 April 2014

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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name

Extracellular signal-regulated ...... sensation in the spinal cord.

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type

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Extracellular signal-regulated ...... sensation in the spinal cord.

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Extracellular signal-regulated ...... sensation in the spinal cord.

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Molecular Brain

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Extracellular signal-regulated ...... sensation in the spinal cord.

@en

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Guan-Yu Jiang

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Jia-Yin Chen

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Ling Zhang

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Ning-Jing Song

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Qing-Xiu Wang

http://www.w3.org/2001/XMLSchema#string

Ying Huang

http://www.w3.org/2001/XMLSchema#string

Yu-Qiang Ding

http://www.w3.org/2001/XMLSchema#string

P2860

Cellular basis of itch sensation

Why we scratch an itch: the molecules, cells and circuits of itch

Itch: scratching more than the surface

The neurobiology of itch

Preserved acute pain and reduced neuropathic pain in mice lacking PKCgamma

Metabotropic glutamate receptor subtypes 1 and 5 are activators of extracellular signal-regulated kinase signaling required for inflammatory pain in mice

The multiple pathways for itch and their interactions with pain

Identification of a novel inhibitor of mitogen-activated protein kinase kinase

Quantitative assessment of tactile allodynia in the rat paw

Chronic itch development in sensory neurons requires BRAF signaling pathways.

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scholarly article

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10.1186/1756-6606-7-25

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7

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2014-04-03T00:00:00Z

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