Podocyte number and density changes during early human life. - Test2 - elhamkhani - TriplyDB

Podocyte number and density changes during early human life.

Podocyte number and density changes during early human life.

http://www.wikidata.org/entity/Q37723409

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Are podocytes motile?Development of the Human Fetal Kidney from Mid to Late Gestation in Male and Female Infants.FSGS as an Adaptive Response to Growth-Induced Podocyte Stress.

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P2860

Podocyte number and density changes during early human life.

http://www.wikidata.org/entity/Q37723409

description

article científic

@ca

article scientifique

@fr

articolo scientifico

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artigo científico

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bilimsel makale

@tr

scientific article published on 27 December 2016

@en

vedecký článok

@sk

vetenskaplig artikel

@sv

videnskabelig artikel

@da

vědecký článek

@cs

name

Podocyte number and density changes during early human life.

@en

Podocyte number and density changes during early human life.

@nl

type

label

Podocyte number and density changes during early human life.

@en

Podocyte number and density changes during early human life.

@nl

prefLabel

Podocyte number and density changes during early human life.

@en

Podocyte number and density changes during early human life.

@nl

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S00467-016-3564-5

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Pediatric Nephrology

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Podocyte number and density changes during early human life.

@en

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Larysa Wickman

http://www.w3.org/2001/XMLSchema#string

Masao Kikuchi

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Raja Rabah

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P2860

Positional cloning uncovers mutations in PLCE1 responsible for a nephrotic syndrome variant that may be reversible

Podometrics as a Potential Clinical Tool for Glomerular Disease Management

Role of the podocyte (and glomerular endothelium) in building the GBM

Unraveling the mechanisms of glomerular ultrafiltration: nephrin, a key component of the slit diaphragm

Genetic testing in steroid-resistant nephrotic syndrome: when and how?

Is nephrogenesis affected by preterm birth? Studies in a non-human primate model.

A single-gene cause in 29.5% of cases of steroid-resistant nephrotic syndrome.

Exploring the genetic basis of early-onset chronic kidney disease.

Rare hereditary COL4A3/COL4A4 variants may be mistaken for familial focal segmental glomerulosclerosis

Accelerated maturation and abnormal morphology in the preterm neonatal kidney.

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s00467-016-3564-5

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823-834

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scholarly article

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10.1007/S00467-016-3564-5

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5

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32

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Roger C Wiggins

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2016-12-27T00:00:00Z

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28028615

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5368211

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