Increased TET1 Expression in Inflammatory Microenvironment of Hyperinsulinemia Enhances the Response of Endometrial Cancer to Estrogen by Epigenetic Modulation of GPER. - Test2 - elhamkhani - TriplyDB

Increased TET1 Expression in Inflammatory Microenvironment of Hyperinsulinemia Enhances the Response of Endometrial Cancer to Estrogen by Epigenetic Modulation of GPER.

Increased TET1 Expression in Inflammatory Microenvironment of Hyperinsulinemia Enhances the Response of Endometrial Cancer to Estrogen by Epigenetic Modulation of GPER.

http://www.wikidata.org/entity/Q37738104

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Estrogen and high-fat diet induced alterations in C57BL/6 mice endometrial transcriptome profile.Insulin resistance and overweight prolonged fertility-sparing treatment duration in endometrial atypical hyperplasia patients.

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Increased TET1 Expression in Inflammatory Microenvironment of Hyperinsulinemia Enhances the Response of Endometrial Cancer to Estrogen by Epigenetic Modulation of GPER.

http://www.wikidata.org/entity/Q37738104

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article científic

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article scientifique

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articolo scientifico

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artigo científico

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bilimsel makale

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scientific article published on 12 March 2017

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vedecký článok

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vetenskaplig artikel

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videnskabelig artikel

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vědecký článek

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Journal of Cancer

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Increased TET1 Expression in I ...... Epigenetic Modulation of GPER.

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Bing-Yi Yang

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Bing-Ying Xie

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Chao Gu

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Cheng-Cheng Ning

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Qiao-Ying Lv

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Wei-Wei Shan

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Xiao-Jun Chen

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Xue-Zhen Luo

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You-Ji Feng

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Zhen-Bo Zhang

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Role of Tet proteins in 5mC to 5hmC conversion, ES-cell self-renewal and inner cell mass specification

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Activation of a positive feedback loop involving IL-6 and aromatase promotes intratumoral 17β-estradiol biosynthesis in endometrial carcinoma microenvironment.

Novel Aspects Concerning the Functional Cross-Talk between the Insulin/IGF-I System and Estrogen Signaling in Cancer Cells.

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Evidence of a Causal Association Between Insulinemia and Endometrial Cancer: A Mendelian Randomization Analysis.

Insulin priming effect on estradiol-induced breast cancer metabolism and growth

Epigenetics, TET proteins, and hypoxia in epithelial-mesenchymal transition and tumorigenesis

Pin1 modulates ERα levels in breast cancer through inhibition of phosphorylation-dependent ubiquitination and degradation.

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scholarly article

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10.7150/JCA.17064

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