Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
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Biogenesis and function of Porphyromonas gingivalis outer membrane vesiclesHost response mechanisms in periodontal diseasesPorphyromonas gingivalis: An Overview of Periodontopathic Pathogen below the Gum LineHow is the development of dental biofilms influenced by the host?Lipoteichoic acid (LTA) and lipopolysaccharides (LPS) from periodontal pathogenic bacteria facilitate oncogenic herpesvirus infection within primary oral cellsBaicalin downregulates Porphyromonas gingivalis lipopolysaccharide-upregulated IL-6 and IL-8 expression in human oral keratinocytes by negative regulation of TLR signaling.Community-wide transcriptome of the oral microbiome in subjects with and without periodontitisAltered oral viral ecology in association with periodontal disease.Interactions of adiponectin and lipopolysaccharide from Porphyromonas gingivalis on human oral epithelial cells.The lipid A phosphate position determines differential host Toll-like receptor 4 responses to phylogenetically related symbiotic and pathogenic bacteria.Inhibitory effects of Ginkgo biloba extract on inflammatory mediator production by Porphyromonas gingivalis lipopolysaccharide in murine macrophages via Nrf-2 mediated heme oxygenase-1 signaling pathways.Application of metagenomics in understanding oral health and disease.Analysis of carbohydrates and glycoconjugates by matrix-assisted laser desorption/ionization mass spectrometry: an update for 2009-2010.Thrombospondin-1 production is enhanced by Porphyromonas gingivalis lipopolysaccharide in THP-1 cells.Functional signatures of oral dysbiosis during periodontitis progression revealed by microbial metatranscriptome analysis.Dual Action of Myricetin on Porphyromonas gingivalis and the Inflammatory Response of Host Cells: A Promising Therapeutic Molecule for Periodontal Diseases.Malva sylvestris Inhibits Inflammatory Response in Oral Human Cells. An In Vitro Infection ModelThe activation of pyrin domain-containing-3 inflammasome depends on lipopolysaccharide from Porphyromonas gingivalis and extracellular adenosine triphosphate in cultured oral epithelial cellsCorrelation of salivary immunoglobulin A against lipopolysaccharide of Porphyromonas gingivalis with clinical periodontal parameters.Do oral bacteria alter the regenerative potential of stem cells? A concise review.Effects of Porphyromonas gingivalis LipopolysaccharideTolerized Monocytes on Inflammatory Responses in Neutrophils.Melatonin Receptor Agonists as the "Perioceutics" Agents for Periodontal Disease through Modulation of Porphyromonas gingivalis Virulence and Inflammatory ResponseA novel class of lipoprotein lipase-sensitive molecules mediates Toll-like receptor 2 activation by Porphyromonas gingivalis.Suppression of T-cell chemokines by Porphyromonas gingivalisDiverse Toll-like receptors mediate cytokine production by Fusobacterium nucleatum and Aggregatibacter actinomycetemcomitans in macrophages.Differential Regulation of Mas-Related G Protein-Coupled Receptor X2-Mediated Mast Cell Degranulation by Antimicrobial Host Defense Peptides and Porphyromonas gingivalis Lipopolysaccharide.Sialylation of Porphyromonas gingivalis LPS and its effect on bacterial-host interactions.Variants of Porphyromonas gingivalis lipopolysaccharide alter lipidation of autophagic protein, microtubule-associated protein 1 light chain 3, LC3.Black tea theaflavins attenuate Porphyromonas gingivalis virulence properties, modulate gingival keratinocyte tight junction integrity and exert anti-inflammatory activity.Antimicrobial Peptides: Mechanisms of Action and Resistance.Identification of Magnolia officinalis L. bark extract as the most potent anti-inflammatory of four plant extracts.Lipopolysaccharide and IL-1β coordinate a synergy on cytokine production by upregulating MyD88 expression in human gingival fibroblasts.Dental follicle progenitor cells responses to Porphyromonas gingivalis LPS.Effect of extracytoplasmic function sigma factors on autoaggregation, hemagglutination, and cell surface properties of Porphyromonas gingivalis.Porphyromonas gingivalis lipopolysaccharide stimulation in human periodontal ligament stem cells: role of epigenetic modifications to the inflammation.Nuclear factor-κB and p38 mitogen-activated protein kinase signaling pathways are critically involved in Porphyromonas gingivalis lipopolysaccharide induction of lipopolysaccharide-binding protein expression in human oral keratinocytes.Porphyromonas gingivalis lipopolysaccharide induces cognitive dysfunction, mediated by neuronal inflammation via activation of the TLR4 signaling pathway in C57BL/6 mice.METTL3 regulates alternative splicing of MyD88 upon the lipopolysaccharide-induced inflammatory response in human dental pulp cells.Porphyromonas gingivalis LPS stimulation downregulates DNMT1, DNMT3a, and JMJD3 gene expression levels in human HaCaT keratinocytes.Porphyromonas gingivalis and its lipopolysaccharide differentially regulate the expression of cathepsin B in endothelial cells.
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Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
description
article científic
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article scientifique
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articolo scientifico
@it
artigo científico
@pt
bilimsel makale
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scientific article published on October 2010
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vedecký článok
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vetenskaplig artikel
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videnskabelig artikel
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vědecký článek
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name
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@en
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@nl
type
label
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@en
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@nl
prefLabel
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@en
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@nl
P2860
P921
P1433
P1476
Contribution of Porphyromonas gingivalis lipopolysaccharide to periodontitis.
@en
P2093
Richard P Darveau
Sumita Jain
P2860
P356
10.1111/J.1600-0757.2009.00333.X
P577
2010-10-01T00:00:00Z