about
Expression of ST3GAL4 leads to SLe(x) expression and induces c-Met activation and an invasive phenotype in gastric carcinoma cellsMatrix metalloproteases as maestros for the dual role of LPS- and IL-10-stimulated macrophages in cancer cell behaviour.E-cadherin-defective gastric cancer cells depend on Laminin to survive and invade.Intricate Macrophage-Colorectal Cancer Cell Communication in Response to Radiation.Ionizing radiation modulates human macrophages towards a pro-inflammatory phenotype preserving their pro-invasive and pro-angiogenic capacities.miR-195 in human primary mesenchymal stromal/stem cells regulates proliferation, osteogenesis and paracrine effect on angiogenesis.Osteopontin-a splice variant is overexpressed in papillary thyroid carcinoma and modulates invasive behavior.Thyroid hormone as a regulator of tumor induced angiogenesis.Interleukin-1B signalling leads to increased survival of gastric carcinoma cells through a CREB-C/EBPβ-associated mechanism.Transcription initiation arising from E-cadherin/CDH1 intron2: a novel protein isoform that increases gastric cancer cell invasion and angiogenesis.CPEB1, a novel gene silenced in gastric cancer: a Drosophila approach.Chromosomal, epigenetic and microRNA-mediated inactivation of LRP1B, a modulator of the extracellular environment of thyroid cancer cells.Inhibition of nociceptive responses after systemic administration of amidated kyotorphinA New Role for Helicobacter pylori Urease: Contributions to Angiogenesis.Participation of mu-opioid, GABA(B), and NK1 receptors of major pain control medullary areas in pathways targeting the rat spinal cord: implications for descending modulation of nociceptive transmission.Noxious-evoked c-fos expression in brainstem neurons immunoreactive for GABAB, mu-opioid and NK-1 receptors.Nociceptive spinal neurons expressing NK1 and GABAB receptors are located in lamina I.DNAJB4 molecular chaperone distinguishes WT from mutant E-cadherin, determining their fate in vitro and in vivo.Dynamic of migration of HSV-1 from a medullary pronociceptive centre: antinociception by overexpression of the preproenkephalin transgene.Microinjection of angiotensin II in the caudal ventrolateral medulla induces hyperalgesia.Opioids modulate pain facilitation from the dorsal reticular nucleus.Guiding morphogenesis in cell-instructive microgels for therapeutic angiogenesis.Inhibitory Effects of Antagonists of Growth Hormone-Releasing Hormone (GHRH) in Thyroid Cancer.Neuronal activation at the spinal cord and medullary pain control centers after joint stimulation: a c-fos study in acute and chronic articular inflammation.Correlation of noxious evoked c-fos expression in areas of the somatosensory system during chronic pain: involvement of spino-medullary and intra-medullary connections.Exosomes secreted by cardiomyocytes subjected to ischaemia promote cardiac angiogenesis.Modelling the tumour microenvironment in long-term microencapsulated 3D co-cultures recapitulates phenotypic features of disease progression.Secondary hyperalgesia in the monoarthritic rat is mediated by GABAB and NK1 receptors of spinal dorsal horn neurons: A behavior and c-fos studyImbalance between the expression of NK1 and GABAB receptors in nociceptive spinal neurons during secondary hyperalgesia: A c-fos study in the monoarthritic ratErratum: Chromosomal, epigenetic and microRNA-mediated inactivation of LRP1B, a modulator of the extracellular environment of thyroid cancer cellsHelicobacter Pylori Targets the EPHA2 Receptor Tyrosine Kinase in Gastric Cells Modulating Key Cellular FunctionsConjugation of the T1 sequence from CCN1 to fibrin hydrogels for therapeutic vascularizationCorrigendum to "Conjugation of the T1 sequence from CCN1 to fibrin hydrogels for therapeutic vascularization" [Mater. Sci. & Eng. C. 104 (2019) 109847]Low Doses of Ionizing Radiation Enhance the Angiogenic Potential of Adipocyte Conditioned Medium
P50
Q31121236-11D28294-34E7-463C-93F3-0476099E585FQ35653181-BBA11F65-5ADF-4D3C-B79C-1D2F4144A706Q36089409-E948BC78-788D-4BA3-A7F0-7F3E7F493A3CQ36101459-90A480ED-DCA2-4221-B141-65DAE90B67EDQ36433820-338624F6-CBA6-4E06-B584-3EE0A584A740Q36729331-B17A13B5-7402-43EA-BF89-957DE2804DB8Q37588399-A5174062-6249-4D5F-95A8-83D8647CF8E3Q37823747-07DA1035-E677-4F46-9422-48CE9A63C334Q38903466-2EDF2B9A-2CA0-43FE-937B-3287D5F5BE24Q39321532-DDF1B60E-6C4A-4008-ADEF-A04EED6329E2Q39448400-8203B8BC-CB9E-4FD7-8DD8-360A0B2226CAQ39634789-D9F56A12-9361-4846-AA6E-3BAE89EB8CA1Q41810823-906AD60B-380A-4006-A647-DDC2246D556BQ42288754-CDD46CCA-3C56-426F-AE0F-B65509F0232AQ44339463-9943A9E2-A3B6-4B9B-B936-1CD05D1FC5B6Q44341370-9DFF1B07-D4F1-4853-8A2D-D8CCE5FB42A8Q44798982-5F4723D7-3F7C-4E3C-A98F-0302AA3F3DF4Q45731458-394176FA-EEF6-4212-A13D-485C5A22A555Q45881655-6BB3E4BB-B662-436A-8FF7-3CAA00AC42A0Q46179273-4BB28100-5ADA-4A26-AD28-DA74B86EF2F9Q46412552-B9F3EBD6-25DF-46E3-95F9-A5B4B63218F8Q46487461-5C0C1739-A959-4CBA-9737-B542CA70ED70Q47765421-7816144B-C247-4F1F-B013-D3B02C193001Q48126807-0B70EA78-14EB-44E3-A40A-71F272B6A3EFQ48392105-DC1C7C39-855C-43AF-86BD-0ABA2CC78C19Q50243885-61E708F5-B92C-4A53-9C34-27DBAAE73F6FQ53385808-F1747C36-AD63-4FF4-B49E-2983D26FB880Q60681744-933D6219-B9B8-4AA7-842A-8FA2AD55C557Q60681745-066D11B0-0975-405E-9C04-3ED5C45CDD9AQ61050529-542530DE-818E-453D-9114-C81FB7C2841BQ89896370-DA401E0E-786A-4B62-AAB4-83C29B51454DQ89992600-DE689B68-B61F-4AB7-BC3F-BEE6B0819C18Q92558571-DFCA3ED0-5118-4630-80B7-05CFAF305449Q92814955-B01B268D-601B-46BF-9D87-B16B31233AE7
P50
description
hulumtuese
@sq
researcher
@en
wetenschapper
@nl
հետազոտող
@hy
name
Marta Pinto
@ast
Marta Pinto
@en
Marta Pinto
@es
Marta Pinto
@nl
Marta Pinto
@sl
type
label
Marta Pinto
@ast
Marta Pinto
@en
Marta Pinto
@es
Marta Pinto
@nl
Marta Pinto
@sl
prefLabel
Marta Pinto
@ast
Marta Pinto
@en
Marta Pinto
@es
Marta Pinto
@nl
Marta Pinto
@sl
P1053
J-2519-2013
P106
P21
P31
P3829
P496
0000-0002-8521-2904