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Tumor Metabolism, the Ketogenic Diet and β-Hydroxybutyrate: Novel Approaches to Adjuvant Brain Tumor TherapyDeterminants of brain cell metabolic phenotypes and energy substrate utilization unraveled with a modeling approachNicotinamide, NAD(P)(H), and Methyl-Group Homeostasis Evolved and Became a Determinant of Ageing Diseases: Hypotheses and Lessons from PellagraUltra-High Mass Resolution MALDI Imaging Mass Spectrometry of Proteins and Metabolites in a Mouse Model of Glioblastoma.Contribution of the Microenvironmental Niche to Glioblastoma Heterogeneity.The ketogenic diet for the treatment of malignant glioma.Molecular mechanisms of fenofibrate-induced metabolic catastrophe and glioblastoma cell death.A unique four-hub protein cluster associates to glioblastoma progression.Japanese encephalitis virus nonstructural protein NS5 interacts with mitochondrial trifunctional protein and impairs fatty acid β-oxidation.The bifunctional autophagic flux by 2-deoxyglucose to control survival or growth of prostate cancer cells.Changes in the expression of proteins associated with aerobic glycolysis and cell migration are involved in tumorigenic ability of two glioma cell linesThe expression pattern of PFKFB3 enzyme distinguishes between induced-pluripotent stem cells and cancer stem cells.EZH2 promotes metabolic reprogramming in glioblastomas through epigenetic repression of EAF2-HIF1α signaling.Clinical aggressiveness of malignant gliomas is linked to augmented metabolism of amino acids.GBM's multifaceted landscape: highlighting regional and microenvironmental heterogeneity.Drug-induced amino acid deprivation as strategy for cancer therapy.Flavopiridol: An Old Drug With New Perspectives? Implication for Development of New Drugs.Metabotropic glutamate receptors in cancer.Autophagic flux in glioblastoma cells.Differentiation of human neuroblastoma cells toward the osteogenic lineage by mTOR inhibitorHypoxia-Mediated Mechanisms Associated with Antiangiogenic Treatment Resistance in Glioblastomas.Metabolic Profiling of IDH Mutation and Malignant Progression in Infiltrating Glioma.The down-regulation of miR-125b in chronic lymphocytic leukemias leads to metabolic adaptation of cells to a transformed state.The antibiotic chloramphenicol may be an effective new agent for inhibiting the growth of multiple myeloma.Multiple endocrine neoplasias: an overview of recent progressLactate is always the end product of glycolysis.Inhibition of HIF1α and PDK Induces Cell Death of Glioblastoma Multiforme.Genetic variants in SLC7A7 are associated with risk of glioma in a Chinese population.Metabolism and glioma therapy.The indoleamine 2,3-dioxygenase inhibitor 1-methyl-tryptophan suppresses mitochondrial function, induces aerobic glycolysis and decreases interleukin-10 production in human lymphocytes.Cancer stem cells (CSCs): metabolic strategies for their identification and eradication.Inflammation-induced DNA methylation of DNA polymerase gamma alters the metabolic profile of colon tumors.Modulation of mitochondrial DNA copy number in a model of glioblastoma induces changes to DNA methylation and gene expression of the nuclear genome in tumours
P2860
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P2860
description
article científic
@ca
article scientifique
@fr
articolo scientifico
@it
artigo científico
@pt
bilimsel makale
@tr
scientific article published on January 2011
@en
vedecký článok
@sk
vetenskaplig artikel
@sv
videnskabelig artikel
@da
vědecký článek
@cs
name
Metabolism and brain cancer.
@en
Metabolism and brain cancer.
@nl
type
label
Metabolism and brain cancer.
@en
Metabolism and brain cancer.
@nl
prefLabel
Metabolism and brain cancer.
@en
Metabolism and brain cancer.
@nl
P2860
P921
P1433
P1476
Metabolism and brain cancer.
@en
P2093
Sueli Mieko Oba Shinjo
Suely Kazue Nagahashi Marie
P2860
P356
10.1590/S1807-59322011001300005
P478
66 Suppl 1
P5008
P577
2011-01-01T00:00:00Z