Disruption of axonal transport in motor neuron diseases.
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A comprehensive review of amyotrophic lateral sclerosisCiliobrevins as tools for studying dynein motor functionA Case for Microtubule Vulnerability in Amyotrophic Lateral Sclerosis: Altered Dynamics During DiseaseInhibition of TFG function causes hereditary axon degeneration by impairing endoplasmic reticulum structure.Exome sequencing of case-unaffected-parents trios reveals recessive and de novo genetic variants in sporadic ALS.Aging, Alzheimer's, and APOE genotype influence the expression and neuronal distribution patterns of microtubule motor protein dynactin-P50Dysregulated expression of death, stress and mitochondrion related genes in the sciatic nerve of presymptomatic SOD1(G93A) mouse model of Amyotrophic Lateral Sclerosis.Golgi fragmentation in amyotrophic lateral sclerosis, an overview of possible triggers and consequences.Mechanisms mediating spinal and bulbar muscular atrophy: investigations into polyglutamine-expanded androgen receptor function and dysfunction.PATHOLOGIES OF AXONAL TRANSPORT IN NEURODEGENERATIVE DISEASESStudy on the dynamic changes in synaptic vesicle-associated protein and axonal transport protein combined with LPS neuroinflammation model.Drosophila melanogaster As a Model Organism to Study RNA Toxicity of Repeat Expansion-Associated Neurodegenerative and Neuromuscular DiseasesRabs, Membrane Dynamics, and Parkinson's Disease.The dynein inhibitor Ciliobrevin D inhibits the bidirectional transport of organelles along sensory axons and impairs NGF-mediated regulation of growth cones and axon branches.DCTN1 gene analysis in Chinese patients with sporadic amyotrophic lateral sclerosis.Identification of 17 Highly Expressed Genes within Mouse Lumbar Spinal Cord Anterior Horn Region from an In-Situ Hybridization Atlas of 3430 Genes: Implications for Motor Neuron Disease.Chaperone-dependent Neurodegeneration: A Molecular Perspective on Therapeutic Intervention.Integrated molecular landscape of amyotrophic lateral sclerosis provides insights into disease etiology.Pharmacological c-Jun NH-Terminal Kinase (JNK) Pathway Inhibition Reduces Severity of Spinal Muscular Atrophy Disease in Mice
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P2860
Disruption of axonal transport in motor neuron diseases.
description
article científic
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article scientifique
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articol științific
@ro
articolo scientifico
@it
artigo científico
@gl
artigo científico
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artigo científico
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artikel ilmiah
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artikull shkencor
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artículo científico
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name
Disruption of axonal transport in motor neuron diseases.
@en
Disruption of axonal transport in motor neuron diseases.
@nl
type
label
Disruption of axonal transport in motor neuron diseases.
@en
Disruption of axonal transport in motor neuron diseases.
@nl
prefLabel
Disruption of axonal transport in motor neuron diseases.
@en
Disruption of axonal transport in motor neuron diseases.
@nl
P2093
P2860
P356
P1476
Disruption of axonal transport in motor neuron diseases.
@en
P2093
Fumiaki Tanaka
Kaori Kawai
Kensuke Ikenaka
Masahisa Katsuno
Shinsuke Ishigaki
P2860
P304
P356
10.3390/IJMS13011225
P407
P50
P5008
P577
2012-01-23T00:00:00Z