The puzzling case of hyperexcitability in amyotrophic lateral sclerosis.
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Modeling ALS with motor neurons derived from human induced pluripotent stem cellsIdentifying the primary site of pathogenesis in amyotrophic lateral sclerosis - vulnerability of lower motor neurons to proximal excitotoxicity.Peripheral challenge with a viral mimic upregulates expression of the complement genes in the hippocampus.F Wave Study in Amyotrophic Lateral Sclerosis: Assessment of Segmental Motoneuronal DysfunctionDissociation of Structural and Functional Integrities of the Motor System in Amyotrophic Lateral Sclerosis and Behavioral-Variant Frontotemporal Dementia.A randomized trial of mexiletine in ALS: Safety and effects on muscle cramps and progressionNeuroimmunity dynamics and the development of therapeutic strategies for amyotrophic lateral sclerosis.Cortical synaptic and dendritic spine abnormalities in a presymptomatic TDP-43 model of amyotrophic lateral sclerosis.Calretinin and Neuropeptide Y interneurons are differentially altered in the motor cortex of the SOD1G93A mouse model of ALS.Amyotrophic lateral sclerosis: a long preclinical period?Motor neuron disease: current management and future prospects.Inhibitory dysfunction in amyotrophic lateral sclerosis: future therapeutic opportunities.Modeling the C9ORF72 repeat expansion mutation using human induced pluripotent stem cells.Impairments in Motor Neurons, Interneurons and Astrocytes Contribute to Hyperexcitability in ALS: Underlying Mechanisms and Paths to Therapy.Synapse Dysfunction of Layer V Pyramidal Neurons Precedes Neurodegeneration in a Mouse Model of TDP-43 Proteinopathies.Patterns of clinical and electrodiagnostic abnormalities in early amyotrophic lateral sclerosis.Electrically evoked multiplet discharges are associated with more marked clinical deterioration in motor neuron disease.Amyotrophic lateral sclerosis-A case report and mechanistic review of the association with toluene and other volatile organic compounds.Impairment of triad conditioned facilitation in amyotrophic lateral sclerosis.Ultra-High Field Proton MR Spectroscopy in Early-Stage Amyotrophic Lateral Sclerosis.Synaptic dysfunction and altered excitability in C9ORF72 ALS/FTD.Nuclear poly(ADP-ribose) activity is a therapeutic target in amyotrophic lateral sclerosis
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P2860
The puzzling case of hyperexcitability in amyotrophic lateral sclerosis.
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article científic
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articolo scientifico
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The puzzling case of hyperexcitability in amyotrophic lateral sclerosis.
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The puzzling case of hyperexcitability in amyotrophic lateral sclerosis.
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prefLabel
The puzzling case of hyperexcitability in amyotrophic lateral sclerosis.
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P2860
P50
P356
P1476
The puzzling case of hyperexcitability in amyotrophic lateral sclerosis
@en
P2093
Matthew C Kiernan
Parvathi Menon
P2860
P356
10.3988/JCN.2013.9.2.65
P577
2013-04-04T00:00:00Z