Dopamine imbalance in Huntington's disease: a mechanism for the lack of behavioral flexibility.
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Corticostriatal Dysfunction in Huntington's Disease: The BasicsHuntingtin Is Required for Neural But Not Cardiac/Pancreatic Progenitor Differentiation of Mouse Embryonic Stem Cells In vitro.A small molecule p75NTR ligand normalizes signalling and reduces Huntington's disease phenotypes in R6/2 and BACHD mice.Developmental exposure to concentrated ambient ultrafine particulate matter air pollution in mice results in persistent and sex-dependent behavioral neurotoxicity and glial activation.Components of the endocannabinoid and dopamine systems are dysregulated in Huntington's disease: analysis of publicly available microarray datasets.Cortical efferents lacking mutant huntingtin improve striatal neuronal activity and behavior in a conditional mouse model of Huntington's diseaseDopaminergic Modulation of Striatal Inhibitory Transmission and Long-Term Plasticity.Interaction without intent: the shape of the social world in Huntington's diseaseInhibition of Excessive Monoamine Oxidase A/B Activity Protects Against Stress-induced Neuronal Death in Huntington Disease.COMT Val158Met Polymorphism Modulates Huntington's Disease Progression.Conservation law for self-paced movementsCorticostriatal synaptic adaptations in Huntington's diseaseThe FOXP2-Driven Network in Developmental Disorders and NeurodegenerationAdvances and challenges in the search for D2 and D3 dopamine receptor-selective compounds.Pridopidine, a dopamine stabilizer, improves motor performance and shows neuroprotective effects in Huntington disease R6/2 mouse model.Adenyl cyclase activator forskolin protects against Huntington's disease-like neurodegenerative disorders.A kinder, gentler dopamine… highlighting dopamine's role in behavioral flexibility.L-theanine, a Component of Green Tea Prevents 3-Nitropropionic Acid (3-NP)-Induced Striatal Toxicity by Modulating Nitric Oxide Pathway.Early pridopidine treatment improves behavioral and transcriptional deficits in YAC128 Huntington disease mice.Disease-modifying effects of ganglioside GM1 in Huntington's disease models.Spatiotemporal Proteomic Profiling of Huntington's Disease Inclusions Reveals Widespread Loss of Protein Function.Modulating Neurotrophin Receptor Signaling as a Therapeutic Strategy for Huntington's Disease.To Do or Not to Do: Dopamine, Affordability and the Economics of Opportunity.Reward feedback accelerates motor learning.Evolution of dopamine receptors: phylogenetic evidence suggests a later origin of the DRD2l and DRD4rs dopamine receptor gene lineages.Environment-dependent striatal gene expression in the BACHD rat model for Huntington disease.Worsening Choreoathetosis in Huntington's Disease with Fluoxetine, Lisdexamfetamine, and Melatonin: A Case Report
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P2860
Dopamine imbalance in Huntington's disease: a mechanism for the lack of behavioral flexibility.
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article científic
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article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
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artículo científico
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Dopamine imbalance in Huntingt ...... ack of behavioral flexibility.
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label
Dopamine imbalance in Huntingt ...... ack of behavioral flexibility.
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prefLabel
Dopamine imbalance in Huntingt ...... ack of behavioral flexibility.
@en
P2093
P2860
P356
P1476
Dopamine imbalance in Huntingt ...... ack of behavioral flexibility.
@en
P2093
Carlos Cepeda
Elizabeth A Wang
Jane Y Chen
Michael S Levine
P2860
P356
10.3389/FNINS.2013.00114
P577
2013-07-04T00:00:00Z