Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
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The Utility of Cerebral Blood Flow as a Biomarker of Preclinical Alzheimer's Disease.Brain imaging of neurovascular dysfunction in Alzheimer's disease.Quantified self and comprehensive geriatric assessment: older adults are able to evaluate their own health and functional status.An age-related shift of resting-state functional connectivity of the subthalamic nucleus: a potential mechanism for compensating motor performance decline in older adultsAlzheimer's disease: relevant molecular and physiopathological events affecting amyloid-β brain balance and the putative role of PPARs.Decreased Default Mode Network connectivity correlates with age-associated structural and cognitive changesJuvenile traumatic brain injury induces long-term perivascular matrix changes alongside amyloid-beta accumulation.Brain amyloid-β burden is associated with disruption of intrinsic functional connectivity within the medial temporal lobe in cognitively normal elderlyCerebral blood flow measured by arterial spin labeling MRI as a preclinical marker of Alzheimer's disease.Characterization of cognitive deficits in spontaneously hypertensive rats, accompanied by brain insulin receptor dysfunction.Third-generation Mineralocorticoid Receptor Antagonists: Why Do We Need a Fourth?Vascular cognitive impairment: Modeling a critical neurologic disease in vitro and in vivo.Higher Brain Perfusion May Not Support Memory Functions in Cognitively Normal Carriers of the ApoE ε4 Allele Compared to Non-Carriers.At the centennial of Michaelis and Menten, competing Michaelis-Menten steps explain effect of GLP-1 on blood-brain transfer and metabolism of glucose.Is Vasomotion in Cerebral Arteries Impaired in Alzheimer's Disease?Calmodulin inhibitor ameliorates cognitive dysfunction via inhibiting nitrosative stress and NLRP3 signaling in mice with bilateral carotid artery stenosis.Netrin-1 Interrupts Amyloid-β Amplification, Increases sAβPPα in vitro and in vivo, and Improves Cognition in a Mouse Model of Alzheimer's Disease.miR-124 Regulates the Expression of BACE1 in the Hippocampus Under Chronic Cerebral Hypoperfusion.Cerebral blood flow and vasoreactivity in aging: an arterial spin labeling studyA Longitudinal Analysis of the Impact of Physical Activity on Neurocognitive Functioning Among HIV-Infected Adults.Regional Gray Matter Atrophy Coexistent with Occipital Periventricular White Matter Hyper Intensities.Divergent Roles of Vascular Burden and Neurodegeneration in the Cognitive Decline of Geriatric Depression Patients and Mild Cognitive Impairment Patients.Impaired hypoxic tolerance in APP23 mice: a dysregulation of neuroprotective globin levels.Cognitive dysfunction in type 2 diabetes patients accompanied with obstructive sleep apnea syndrome.Rosuvastatin Attenuates High-Salt and Cholesterol Diet Induced Neuroinflammation and Cognitive Impairment via Preventing Nuclear Factor KappaB Pathway.Astrocyte and Alzheimer's disease.Are Major Dementias Triggered by Poor Blood Flow to the Brain? Theoretical Considerations.Mediators of episodic memory decay across the adult life span.Modulation of mitochondrial bioenergetics as a therapeutic strategy in Alzheimer's disease.Stimulus-evoked changes in cerebral vessel diameter: A study in healthy humans.
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Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
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article científic
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article scientifique
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articol științific
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articolo scientifico
@it
artigo científico
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artigo científico
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artigo científico
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Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
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type
label
Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
@en
prefLabel
Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
@en
P2093
P2860
P1476
Vascular cognitive impairment, dementia, aging and energy demand. A vicious cycle.
@en
P2093
A Popa-Wagner
Ana-Maria Buga
D Muresanu
P2860
P2888
P304
P356
10.1007/S00702-013-1129-3
P478
122 Suppl 1
P577
2013-12-14T00:00:00Z