Anandamide in primary sensory neurons: too much of a good thing?

Anandamide in primary sensory neurons: too much of a good thing?

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http://www.wikidata.org/entity/Q38185073
Are sensory TRP channels biological alarms for lipid peroxidation?Endovanilloids are potential activators of the trigeminovascular nocisensor complexActivation of CB1 inhibits NGF-induced sensitization of TRPV1 in adult mouse afferent neurons.Spatial Distribution of the Cannabinoid Type 1 and Capsaicin Receptors May Contribute to the Complexity of Their Crosstalk.Peripheral inflammation affects modulation of nociceptive synaptic transmission in the spinal cord induced by N-arachidonoylphosphatidylethanolamine.Inflammation of peripheral tissues and injury to peripheral nerves induce differing effects in the expression of the calcium-sensitive N-arachydonoylethanolamine-synthesizing enzyme and related molecules in rat primary sensory neurons.Alterations in the anandamide metabolism in the development of neuropathic pain.
P2860

Anandamide in primary sensory neurons: too much of a good thing?

Item
http://www.wikidata.org/entity/Q38185073
description
article científic
@ca
article scientifique
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articol științific
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articolo scientifico
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artigo científico
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artigo científico
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artigo científico
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artikel ilmiah
@id
artikull shkencor
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artículo científico
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name
Anandamide in primary sensory neurons: too much of a good thing?
@en
type
label
Anandamide in primary sensory neurons: too much of a good thing?
@en
prefLabel
Anandamide in primary sensory neurons: too much of a good thing?
@en
P1433
P1476
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921
P356
P1433
P1476
Anandamide in primary sensory neurons: too much of a good thing?
@en
P2093
P2860
P304
P31
P356
P407
P433
P478
P50
P577
P698
P921