VP4 differentially regulates TRAF2 signaling, disengaging JNK activation while directing NF-kappa B to effect rotavirus-specific cellular responses.
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Modulation of tumor necrosis factor by microbial pathogens.Vaccinia virus protein A52R activates p38 mitogen-activated protein kinase and potentiates lipopolysaccharide-induced interleukin-10Rotavirus nonstructural protein 1 suppresses virus-induced cellular apoptosis to facilitate viral growth by activating the cell survival pathways during early stages of infectionThe rhesus rotavirus VP4 sialic acid binding domain has a galectin fold with a novel carbohydrate binding site.Tumor necrosis factor receptor-associated factors (TRAFs)Differential infection of polarized epithelial cell lines by sialic acid-dependent and sialic acid-independent rotavirus strains.High-resolution molecular and antigen structure of the VP8* core of a sialic acid-independent human rotavirus strain.VLA-2 (alpha2beta1) integrin promotes rotavirus entry into cells but is not necessary for rotavirus attachment.Rotavirus activates JNK and p38 signaling pathways in intestinal cells, leading to AP-1-driven transcriptional responses and enhanced virus replicationNF-kappaB and virus infection: who controls whom.The poxvirus protein A52R targets Toll-like receptor signaling complexes to suppress host defenseAnti-rotaviral effects of Glycyrrhiza uralensis extract in piglets with rotavirus diarrhea.Leukemia virus long terminal repeat activates NFkappaB pathway by a TLR3-dependent mechanism.Poxviral protein A52 stimulates p38 mitogen-activated protein kinase (MAPK) activation by causing tumor necrosis factor receptor-associated factor 6 (TRAF6) self-association leading to transforming growth factor β-activated kinase 1 (TAK1) recruitmeInhibition of rotavirus ECwt infection in ICR suckling mice by N-acetylcysteine, peroxisome proliferator-activated receptor gamma agonists and cyclooxygenase-2 inhibitors.The influenza A virus NS1 protein inhibits activation of Jun N-terminal kinase and AP-1 transcription factors.Geometric mismatches within the concentric layers of rotavirus particles: a potential regulatory switch of viral particle transcription activity.Inhibition of cyclooxygenase activity reduces rotavirus infection at a postbinding step.Inhibition of tumor necrosis factor (TNF) signal transduction by the adenovirus group C RID complex involves downregulation of surface levels of TNF receptor 1.Hyperphosphorylation of the rotavirus NSP5 protein is independent of serine 67, [corrected] NSP2, or [corrected] the intrinsic insolubility of NSP5 is regulated by cellular phosphatases.Rhesus rotavirus VP6 regulates ERK-dependent calcium influx in cholangiocytes.A parapoxviral virion protein targets the retinoblastoma protein to inhibit NF-κB signaling.
P2860
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P2860
VP4 differentially regulates TRAF2 signaling, disengaging JNK activation while directing NF-kappa B to effect rotavirus-specific cellular responses.
description
2001 nî lūn-bûn
@nan
2001年の論文
@ja
2001年論文
@yue
2001年論文
@zh-hant
2001年論文
@zh-hk
2001年論文
@zh-mo
2001年論文
@zh-tw
2001年论文
@wuu
2001年论文
@zh
2001年论文
@zh-cn
name
VP4 differentially regulates T ...... s-specific cellular responses.
@en
type
label
VP4 differentially regulates T ...... s-specific cellular responses.
@en
prefLabel
VP4 differentially regulates T ...... s-specific cellular responses.
@en
P2093
P2860
P356
P1476
VP4 differentially regulates T ...... s-specific cellular responses.
@en
P2093
E Geimonen
E R Mackow
J Nazarova
R LaMonica
P2860
P304
19889-19896
P356
10.1074/JBC.M100499200
P407
P577
2001-03-21T00:00:00Z