Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.
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PPAR-gamma: Therapeutic Potential for Multiple SclerosisTranscription factor IIA mutations show activator-specific defects and reveal a IIA function distinct from stimulation of TBP-DNA bindingDevelopmental effects of ectopic expression of the glucocorticoid receptor DNA binding domain are alleviated by an amino acid substitution that interferes with homeodomain bindingCorticosteroid-resistant bronchial asthma is associated with increased c-fos expression in monocytes and T lymphocytesNF-IL6 and AP-1 cooperatively modulate the activation of the TSG-6 gene by tumor necrosis factor alpha and interleukin-1Selective binding of steroid hormone receptors to octamer transcription factors determines transcriptional synergism at the mouse mammary tumor virus promoterInduction of the E-selectin promoter by interleukin 1 and tumour necrosis factor alpha, and inhibition by glucocorticoidsCyclopentenone prostaglandins: new insights on biological activities and cellular targetsMolecular determinants of NOTCH4 transcription in vascular endothelium.Protection of neurons and microglia against ethanol in a mouse model of fetal alcohol spectrum disorders by peroxisome proliferator-activated receptor-γ agonists.Effects of peroxisome proliferator-activated receptor-gamma agonists on central nervous system inflammation.Molecular aspects of glucocorticoid hormone action in rheumatoid arthritis.Autocrine growth and anchorage independence: two complementing Jun-controlled genetic programs of cellular transformation.Energy transfer analysis of Fos-Jun dimerization and DNA bindingBudesonide epimer R or dexamethasone selectively inhibit platelet-activating factor-induced or interleukin 1beta-induced DNA binding activity of cis-acting transcription factors and cyclooxygenase-2 gene expression in human epidermal keratinocytes.Regulation of the cd38 promoter in human airway smooth muscle cells by TNF-alpha and dexamethasoneTranscriptional repression of the interleukin-2 gene by vitamin D3: direct inhibition of NFATp/AP-1 complex formation by a nuclear hormone receptorHormone-independent repression of AP-1-inducible collagenase promoter activity by glucocorticoid receptors.Antagonism between Nur77 and glucocorticoid receptor for control of transcription.Incoherent feed-forward regulatory logic underpinning glucocorticoid receptor action.Therapeutic implications of targeting energy metabolism in breast cancer.S. aureus-dependent microglial activation is selectively attenuated by the cyclopentenone prostaglandin 15-deoxy-Delta12,14- prostaglandin J2 (15d-PGJ2)Fos and Jun repress transcription activation by NF-IL6 through association at the basic zipper region.Proteasome regulation of activation-induced T cell death.Glucocorticoid regulation of CD38 expression in human airway smooth muscle cells: role of dual specificity phosphatase 1.Restriction to Fos family members of Trip6-dependent coactivation and glucocorticoid receptor-dependent trans-repression of activator protein-1.Interaction of glucocorticoid receptor (GR) with estrogen receptor (ER) α and activator protein 1 (AP1) in dexamethasone-mediated interference of ERα activityGlucocorticoid receptor and breast cancer.Negative regulation of a special, double AP-1 consensus element in the vimentin promoter: interference by the retinoic acid receptor.Transcriptional inhibition of stromelysin by interferon-gamma in normal human fibroblasts is mediated by the AP-1 domain.Androgen receptor-associated protein complex binds upstream of the androgen-responsive elements in the promoters of human prostate-specific antigen and kallikrein 2 genes.The leucine domain of the visna virus Tat protein mediates targeting to an AP-1 site in the viral long terminal repeat.Characterization of mechanisms involved in transrepression of NF-kappa B by activated glucocorticoid receptors.Glucocorticoid resistance in asthma.Mechanisms of glucocorticoid action in bone cells.Thyroid hormone receptor can modulate retinoic acid-mediated axis formation in frog embryogenesis.A distinct modulating domain in glucocorticoid receptor monomers in the repression of activity of the transcription factor AP-1.Negative regulation of transcription in eukaryotes.Differential modes of regulation of interleukin-1beta expression by extracellular matrices.The interleukin-1 type 2 receptor gene displays immediate early gene responsiveness in glucocorticoid-stimulated human epidermal keratinocytes.
P2860
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P2860
Fos is a preferential target of glucocorticoid receptor inhibition of AP-1 activity in vitro.
description
1993 nî lūn-bûn
@nan
1993年の論文
@ja
1993年学术文章
@wuu
1993年学术文章
@zh-cn
1993年学术文章
@zh-hans
1993年学术文章
@zh-my
1993年学术文章
@zh-sg
1993年學術文章
@yue
1993年學術文章
@zh
1993年學術文章
@zh-hant
name
Fos is a preferential target o ...... ion of AP-1 activity in vitro.
@en
type
label
Fos is a preferential target o ...... ion of AP-1 activity in vitro.
@en
prefLabel
Fos is a preferential target o ...... ion of AP-1 activity in vitro.
@en
P2093
P2860
P356
P1476
Fos is a preferential target o ...... ion of AP-1 activity in vitro.
@en
P2093
P2860
P304
P356
10.1128/MCB.13.6.3782
P407
P577
1993-06-01T00:00:00Z