trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
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Human cytomegalovirus uracil DNA glycosylase associates with ppUL44 and accelerates the accumulation of viral DNA.Herpesvirus Late Gene Expression: A Viral-Specific Pre-initiation Complex Is KeyEpstein-Barr virus and host cell methylation: regulation of latency, replication and virus reactivationInhibition of Epstein-Barr virus replication by a benzimidazole L-riboside: novel antiviral mechanism of 5, 6-dichloro-2-(isopropylamino)-1-beta-L-ribofuranosyl-1H-benzimidazole.Mutual inhibition between Kaposi's sarcoma-associated herpesvirus and Epstein-Barr virus lytic replication initiators in dually-infected primary effusion lymphomaIdentification and Function of MicroRNAs Encoded by HerpesvirusesAlphaherpesvirus origin-binding protein homolog encoded by human herpesvirus 6B, a betaherpesvirus, binds to nucleotide sequences that are similar to ori regions of alphaherpesvirusesMta has properties of an RNA export protein and increases cytoplasmic accumulation of Epstein-Barr virus replication gene mRNA.Identification of Epstein-Barr Virus Replication Proteins in Burkitt's Lymphoma Cells.A role for the nucleosome assembly proteins TAF-Iβ and NAP1 in the activation of BZLF1 expression and Epstein-Barr virus reactivationAP-1 homolog BZLF1 of Epstein-Barr virus has two essential functions dependent on the epigenetic state of the viral genome.A subset of replication proteins enhances origin recognition and lytic replication by the Epstein-Barr virus ZEBRA protein.CpG-methylation regulates a class of Epstein-Barr virus promoters.The Epstein-Barr virus (EBV) SM protein enhances pre-mRNA processing of the EBV DNA polymerase transcript.The Epstein-Barr virus lytic transactivator Zta interacts with the helicase-primase replication proteins.The Epstein-Barr virus BZLF1 protein interacts physically and functionally with the histone acetylase CREB-binding protein.Arsenic trioxide inhibits EBV reactivation and promotes cell death in EBV-positive lymphoma cells.The human cytomegalovirus IE2 and UL112-113 proteins accumulate in viral DNA replication compartments that initiate from the periphery of promyelocytic leukemia protein-associated nuclear bodies (PODs or ND10)Origin-independent assembly of Kaposi's sarcoma-associated herpesvirus DNA replication compartments in transient cotransfection assays and association with the ORF-K8 protein and cellular PML.Spontaneous activation of the lytic cycle in cells infected with a recombinant Kaposi's sarcoma-associated virus.Kaposi's sarcoma-associated herpesvirus K-bZIP represses gene transcription via SUMO modification.Multiple layers of cooperativity regulate enhanceosome-responsive RNA polymerase II transcription complex assembly.Mechanism for specificity by HMG-1 in enhanceosome assemblyInteraction of Kaposi's sarcoma-associated herpesvirus ORF6 protein with single-stranded DNA.Uracil DNA glycosylase BKRF3 contributes to Epstein-Barr virus DNA replication through physical interactions with proteins in viral DNA replication complex.Evolutionary aspects of oncogenic herpesvirusesEpstein-Barr virus mRNA export factor EB2 is essential for intranuclear capsid assembly and production of gp350Kaposi's sarcoma-associated herpesvirus-encoded latency-associated nuclear antigen inhibits lytic replication by targeting Rta: a potential mechanism for virus-mediated control of latency.The Epstein-Barr virus BZLF1 protein inhibits tumor necrosis factor receptor 1 expression through effects on cellular C/EBP proteinsEpstein-Barr virus mRNA export factor EB2 is essential for production of infectious virus.Use of adenovirus vectors expressing Epstein-Barr virus (EBV) immediate-early protein BZLF1 or BRLF1 to treat EBV-positive tumors.BZLF1 governs CpG-methylated chromatin of Epstein-Barr Virus reversing epigenetic repression.CCAAT/enhancer binding protein alpha interacts with ZTA and mediates ZTA-induced p21(CIP-1) accumulation and G(1) cell cycle arrest during the Epstein-Barr virus lytic cycle.Latent membrane protein 1 inhibits Epstein-Barr virus lytic cycle induction and progress via different mechanisms.The importance of epigenetic alterations in the development of epstein-barr virus-related lymphomas.Epstein-Barr virus late gene transcription depends on the assembly of a virus-specific preinitiation complex.The Kaposi's sarcoma-associated herpesvirus ORF6 DNA binding protein forms long DNA-free helical protein filamentsKaposi's sarcoma-associated herpesvirus lytic origin (ori-Lyt)-dependent DNA replication: identification of the ori-Lyt and association of K8 bZip protein with the originThe latency-associated nuclear antigen homolog of herpesvirus saimiri inhibits lytic virus replication.ORF18 is a transfactor that is essential for late gene transcription of a gammaherpesvirus.
P2860
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P2860
trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
description
1992 nî lūn-bûn
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1992年の論文
@ja
1992年学术文章
@wuu
1992年学术文章
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1992年学术文章
@zh-hans
1992年学术文章
@zh-my
1992年学术文章
@zh-sg
1992年學術文章
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1992年學術文章
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1992年學術文章
@zh-hant
name
trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
@en
type
label
trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
@en
prefLabel
trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
@en
P2093
P2860
P1433
P1476
trans-acting requirements for replication of Epstein-Barr virus ori-Lyt.
@en
P2093
E D Fixman
G S Hayward
S D Hayward
P2860
P304
P407
P577
1992-08-01T00:00:00Z