Autophagy, a double-edged sword in anti-angiogenesis therapy. - Test2 - elhamkhani - TriplyDB

Autophagy, a double-edged sword in anti-angiogenesis therapy.

Autophagy, a double-edged sword in anti-angiogenesis therapy.

http://www.wikidata.org/entity/Q38682453

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Modulation of Autophagy by Sorafenib: Effects on Treatment Response Cell apoptosis, autophagy and necroptosis in osteosarcoma treatment Minimally invasive pancreatic cancer surgery: What is the current evidence?Overactivated autophagy contributes to steroid-induced avascular necrosis of the femoral head.ERAS protocol in laparoscopic surgery for colonic versus rectal carcinoma: are there differences in short-term outcomes?A Systematic Review of the Immune-Regulating and Anticancer Activities of Pseudolaric Acid B.The role of autophagy in hepatocellular carcinoma: friend or foe.GADD45β induction by S-adenosylmethionine inhibits hepatocellular carcinoma cell proliferation during acute ischemia-hypoxia.Chloroquine potentiates the anticancer effect of sunitinib on renal cell carcinoma by inhibiting autophagy and inducing apoptosis.Autophagy: a potential target for the treatment of intraocular neovascularization.Crocin induces autophagic apoptosis in hepatocellular carcinoma by inhibiting Akt/mTOR activity.Manipulation of autophagy: a novelly potential therapeutic strategy for retinal neovascularization.Long-Zhi Decoction Medicated Serum Promotes Angiogenesis in Human Umbilical Vein Endothelial Cells Based on Autophagy.Inhibition of retinal angiogenesis by gold nanoparticles inducing autophagy

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Autophagy, a double-edged sword in anti-angiogenesis therapy.

http://www.wikidata.org/entity/Q38682453

description

2015 nî lūn-bûn

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2015年の論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年論文

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2015年论文

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2015年论文

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2015年论文

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name

Autophagy, a double-edged sword in anti-angiogenesis therapy.

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Autophagy, a double-edged sword in anti-angiogenesis therapy.

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Autophagy, a double-edged sword in anti-angiogenesis therapy.

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Medical Oncology

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Autophagy, a double-edged sword in anti-angiogenesis therapy.

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Guoping Sun

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Hua Wang

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Jiatao Liu

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Lulu Fan

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P2860

Anti-PlGF inhibits growth of VEGF(R)-inhibitor-resistant tumors without affecting healthy vessels

Cytosolic FoxO1 is essential for the induction of autophagy and tumour suppressor activity

Beclin 1, an autophagy gene essential for early embryonic development, is a haploinsufficient tumor suppressor

Hypoxia-induced lysyl oxidase is a critical mediator of bone marrow cell recruitment to form the premetastatic niche

Modes of resistance to anti-angiogenic therapy

Antiangiogenic therapy elicits malignant progression of tumors to increased local invasion and distant metastasis

Promotion of tumorigenesis by heterozygous disruption of the beclin 1 autophagy gene

Reactive oxygen species are essential for autophagy and specifically regulate the activity of Atg4

CCI-779 inhibits rhabdomyosarcoma xenograft growth by an antiangiogenic mechanism linked to the targeting of mTOR/Hif-1alpha/VEGF signaling

Signaling unmasked: Autophagy and catalase promote programmed cell death

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