The β2-Adrenoceptor Agonist Terbutaline Stimulates Angiogenesis via Akt and ERK Signaling.
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Restricting the induction of NGF in ovarian stroma engenders selective follicular activation through the mTOR signaling pathway.Developmental Neurotoxicity Resulting from Pharmacotherapy of Preterm Labor, Modeled In Vitro: Terbutaline and Dexamethasone, Separately and Together.HDAC3 Inhibition Promotes Alternative Activation of Macrophages but Does Not Affect Functional Recovery after Spinal Cord Injury
P2860
The β2-Adrenoceptor Agonist Terbutaline Stimulates Angiogenesis via Akt and ERK Signaling.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
The β2-Adrenoceptor Agonist Te ...... sis via Akt and ERK Signaling.
@en
type
label
The β2-Adrenoceptor Agonist Te ...... sis via Akt and ERK Signaling.
@en
prefLabel
The β2-Adrenoceptor Agonist Te ...... sis via Akt and ERK Signaling.
@en
P2093
P2860
P356
P1476
The β2-Adrenoceptor Agonist Terbutaline Stimulates Angiogenesis via Akt and ERK Signaling
@en
P2093
Lauren Kusters
Nathalie Geurts
Stefanie Lemmens
P2860
P304
P356
10.1002/JCP.25483
P577
2016-07-20T00:00:00Z