20-HETE attenuates the response of glucose-stimulated insulin secretion through the AKT/GSK-3β/Glut2 pathway.

about

20-HETE promotes glucose-stimulated insulin secretion in an autocrine manner through FFAR1.20-HETE: Hypertension and Beyond

P2860

Q49549662-F6A606CD-2A49-4DAE-AB41-EC07AD6E7555 Q57210352-A34EC6A7-5788-4ADD-9A56-970A97FEAF69

P2860

20-HETE attenuates the response of glucose-stimulated insulin secretion through the AKT/GSK-3β/Glut2 pathway.

Item

http://www.wikidata.org/entity/Q38736558

description

2016 nî lūn-bûn

@nan

2016年の論文

@ja

2016年論文

@yue

2016年論文

@zh-hant

2016年論文

@zh-hk

2016年論文

@zh-mo

2016年論文

@zh-tw

2016年论文

@wuu

2016年论文

@zh

2016年论文

@zh-cn

name

20-HETE attenuates the respons ...... the AKT/GSK-3β/Glut2 pathway.

@en

type

Item

label

20-HETE attenuates the respons ...... the AKT/GSK-3β/Glut2 pathway.

@en

prefLabel

20-HETE attenuates the respons ...... the AKT/GSK-3β/Glut2 pathway.

@en

P1476

20-HETE attenuates the respons ...... the AKT/GSK-3β/Glut2 pathway.

@en

P2093

Bijun Zhang

http://www.w3.org/2001/XMLSchema#string

Guangrui Lai

http://www.w3.org/2001/XMLSchema#string

Jingjing Wu

http://www.w3.org/2001/XMLSchema#string

Ru Sun

http://www.w3.org/2001/XMLSchema#string

Runhong Xu

http://www.w3.org/2001/XMLSchema#string

Xianghong Yang

http://www.w3.org/2001/XMLSchema#string

Yafei Qi

http://www.w3.org/2001/XMLSchema#string

Yanyan Zhao

http://www.w3.org/2001/XMLSchema#string

P2860

Free fatty acids increase cytosolic free calcium and stimulate insulin secretion from beta-cells through activation of GPR40.

Requirement for glycogen synthase kinase-3beta in cell survival and NF-kappaB activation

Early diabetes and abnormal postnatal pancreatic islet development in mice lacking Glut-2

Cyclical and alternating infusions of glucose and intralipid in rats inhibit insulin gene expression and Pdx-1 binding in islets.

P-450 metabolites of arachidonic acid in the control of cardiovascular function

A feat of metabolic proportions: Pdx1 orchestrates islet development and function in the maintenance of glucose homeostasis.

20-HETE induces hyperglycemia through the cAMP/PKA-PhK-GP pathway.

Inhibition or deletion of soluble epoxide hydrolase prevents hyperglycemia, promotes insulin secretion, and reduces islet apoptosis.

Acute inhibition of PI3K-PDK1-Akt pathway potentiates insulin secretion through upregulation of newcomer granule fusions in pancreatic β-cells.

A GSK-3/TSC2/mTOR pathway regulates glucose uptake and GLUT1 glucose transporter expression

P2888

s12020-016-1031-5

P304

371-382

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1007/S12020-016-1031-5

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P577

2016-08-27T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

27568038

http://www.w3.org/2001/XMLSchema#string

20-HETE attenuates the response of glucose-stimulated insulin secretion through the AKT/GSK-3β/Glut2 pathway.

about

P2860

P2860

20-HETE attenuates the response of glucose-stimulated insulin secretion through the AKT/GSK-3β/Glut2 pathway.

description

name

type

label

prefLabel

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P577

P698

P356

P1433

P1476

P2093

P2860

P2888

P304

P31

P356

P433

P478

P577

P698