Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
about
Upregulation of miR-665 promotes apoptosis and colitis in inflammatory bowel disease by repressing the endoplasmic reticulum stress components XBP1 and ORMDL3.The Unfolded Protein Response in the Immune Cell Development: Putting the Caretaker in the Driving Seat.Hydrogen-rich water protects against inflammatory bowel disease in mice by inhibiting endoplasmic reticulum stress and promoting heme oxygenase-1 expression.CHOP-induced loss of intestinal epithelial stemness contributes to bile duct ligation-induced cholestatic liver injury.Biopsy-derived Intestinal Epithelial Cell Cultures for Pathway-based Stratification of Patients With Inflammatory Bowel Disease.Severe Burn-Induced Intestinal Epithelial Barrier Dysfunction Is Associated With Endoplasmic Reticulum Stress and Autophagy in Mice.FKBP11 protects intestinal epithelial cells against inflammation‑induced apoptosis via the JNK‑caspase pathway in Crohn's disease
P2860
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P2860
Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
@en
type
label
Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
@en
prefLabel
Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
@en
P2860
P1476
Epithelial ER Stress in Crohn's Disease and Ulcerative Colitis.
@en
P2093
Stewart S Cao
P2860
P304
P356
10.1097/MIB.0000000000000660
P577
2016-03-04T00:00:00Z