Acetylation and succinylation contribute to maturational alterations in energy metabolism in the newborn heart.
about
Acetylation of mitochondrial proteins by GCN5L1 promotes enhanced fatty acid oxidation in the heart.Mechanisms of transcription factor acetylation and consequences in hearts.GCN5L1 modulates cross-talk between mitochondria and cell signaling to regulate FoxO1 stability and gluconeogenesis.Inflexibility of AMPK-mediated metabolic reprogramming in mitochondrial disease.GCN5L1/BLOS1 Links Acetylation, Organelle Remodeling, and Metabolism.Acetylation contributes to hypertrophy-caused maturational delay of cardiac energy metabolism.The protein acetylase GCN5L1 modulates hepatic fatty acid oxidation activity via acetylation of the mitochondrial β-oxidation enzyme HADHARole of the Pyruvate Dehydrogenase Complex in Metabolic Remodeling: Differential Pyruvate Dehydrogenase Complex Functions in Metabolism
P2860
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P2860
Acetylation and succinylation contribute to maturational alterations in energy metabolism in the newborn heart.
description
2016 nî lūn-bûn
@nan
2016年の論文
@ja
2016年論文
@yue
2016年論文
@zh-hant
2016年論文
@zh-hk
2016年論文
@zh-mo
2016年論文
@zh-tw
2016年论文
@wuu
2016年论文
@zh
2016年论文
@zh-cn
name
Acetylation and succinylation ...... tabolism in the newborn heart.
@en
type
label
Acetylation and succinylation ...... tabolism in the newborn heart.
@en
prefLabel
Acetylation and succinylation ...... tabolism in the newborn heart.
@en
P2093
P2860
P1476
Acetylation and succinylation ...... etabolism in the newborn heart
@en
P2093
Arata Fukushima
Cory S Wagg
Gary D Lopaschuk
Ivan M Rebeyka
Liyan Zhang
Osama Abo Alrob
Sonia Rawat
Tariq Altamimi
P2860
P304
P356
10.1152/AJPHEART.00900.2015
P577
2016-06-03T00:00:00Z