Overexpression of HOX genes is prevalent in Ewing sarcoma and is associated with altered epigenetic regulation of developmental transcription programs.
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The second European interdisciplinary Ewing sarcoma research summit--A joint effort to deconstructing the multiple layers of a complex diseaseLoss of Ewing sarcoma EWS allele promotes tumorigenesis by inducing chromosomal instability in zebrafish.Tumorigenicity of Ewing sarcoma is critically dependent on the trithorax proteins MLL1 and menin.Promoter Methylation Analysis Reveals That KCNA5 Ion Channel Silencing Supports Ewing Sarcoma Cell Proliferation.The posterior HOXD locus: Its contribution to phenotype and malignancy of Ewing sarcoma.The histone demethylase KDM3A, and its downstream target MCAM, promote Ewing Sarcoma cell migration and metastasis.A bivalent promoter contributes to stress-induced plasticity of CXCR4 in Ewing sarcomaHigh EZH2 expression is correlated to metastatic disease in pediatric soft tissue sarcomas.Identification of genetic variants in pharmacokinetic genes associated with Ewing Sarcoma treatment outcome.The Jumonji-domain histone demethylase inhibitor JIB-04 deregulates oncogenic programs and increases DNA damage in Ewing Sarcoma, resulting in impaired cell proliferation and survival, and reduced tumor growth
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Overexpression of HOX genes is prevalent in Ewing sarcoma and is associated with altered epigenetic regulation of developmental transcription programs.
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Overexpression of HOX genes is ...... mental transcription programs.
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Overexpression of HOX genes is ...... mental transcription programs.
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Overexpression of HOX genes is ...... mental transcription programs.
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Overexpression of HOX genes is ...... pmental transcription programs
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Ashley Harris
Brian Magnuson
Cornelia von Levetzow
Elizabeth R Lawlor
Laurie K Svoboda
Mats Ljungman
Natashay J Bailey
Raphaela Schwentner
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10.4161/15592294.2014.988048
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2014-12-01T00:00:00Z