Effects of copper overload in P19 neurons: impairment of glutathione redox homeostasis and crosstalk between caspase and calpain protease systems in ROS-induced apoptosis.
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Ceramides in Alzheimer's Disease: Key Mediators of Neuronal Apoptosis Induced by Oxidative Stress and Aβ AccumulationMembrane of Candida albicans as a target of berberine.Chemosensitivity of U251 Cells to the Co-treatment of D-Penicillamine and Copper: Possible Implications on Wilson Disease Patients.Oxidative damage induced by copper in mouse primary hepatocytes by single-cell analysis.High copper concentrations produce genotoxicity and cytotoxicity in bovine cumulus cells.
P2860
Effects of copper overload in P19 neurons: impairment of glutathione redox homeostasis and crosstalk between caspase and calpain protease systems in ROS-induced apoptosis.
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
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2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
Effects of copper overload in ...... tems in ROS-induced apoptosis.
@en
type
label
Effects of copper overload in ...... tems in ROS-induced apoptosis.
@en
prefLabel
Effects of copper overload in ...... tems in ROS-induced apoptosis.
@en
P2093
P2860
P1433
P1476
Effects of copper overload in ...... tems in ROS-induced apoptosis.
@en
P2093
Josipa Vlainić
Julija Erhardt
Maja Jazvinšćak Jembrek
Nada Oršolić
Vedrana Radovanović
P2860
P2888
P304
P356
10.1007/S10534-014-9792-X
P577
2014-09-14T00:00:00Z
P6179
1029947100