Deficiency of the Angiotensinase Aminopeptidase A Increases Susceptibility to Glomerular Injury.

about

Deficiency of the Angiotensinase Aminopeptidase A Increases Susceptibility to Glomerular Injury.

Item

http://www.wikidata.org/entity/Q38959449

description

2017 nî lūn-bûn

@nan

2017年の論文

@ja

2017年論文

@yue

2017年論文

@zh-hant

2017年論文

@zh-hk

2017年論文

@zh-mo

2017年論文

@zh-tw

2017年论文

@wuu

2017年论文

@zh

2017年论文

@zh-cn

name

Deficiency of the Angiotensina ...... tibility to Glomerular Injury.

@en

type

Item

label

Deficiency of the Angiotensina ...... tibility to Glomerular Injury.

@en

prefLabel

Deficiency of the Angiotensina ...... tibility to Glomerular Injury.

@en

P1476

Deficiency of the Angiotensina ...... tibility to Glomerular Injury.

@en

P2093

Carl Atkinson

http://www.w3.org/2001/XMLSchema#string

Deepak Nihalani

http://www.w3.org/2001/XMLSchema#string

Evelyn T Bruner

http://www.w3.org/2001/XMLSchema#string

Jessalyn Rodgers

http://www.w3.org/2001/XMLSchema#string

Juan Carlos Q Velez

http://www.w3.org/2001/XMLSchema#string

Megan P Hicks

http://www.w3.org/2001/XMLSchema#string

Michael G Janech

http://www.w3.org/2001/XMLSchema#string

Milos N Budisavljevic

http://www.w3.org/2001/XMLSchema#string

Wayne R Fitzgibbon

http://www.w3.org/2001/XMLSchema#string

P2860

Motor protein Myo1c is a podocyte protein that facilitates the transport of slit diaphragm protein Neph1 to the podocyte membrane

Evaluation of angiotensin-converting enzyme (ACE), its homologue ACE2 and neprilysin in angiotensin peptide metabolism

Aminopeptidase A: a nephritogenic target antigen of nephrotoxic serum.

Angiotensin II type 1 receptor overexpression in podocytes induces glomerulosclerosis in transgenic rats.

Increased glomerular capillary pressure and size mediate glomerulosclerosis in SHR juxtamedullary cortex.

Aminopeptidase-A. I. CDNA cloning and expression and localization in rat tissues.

Characterization of renin-angiotensin system enzyme activities in cultured mouse podocytes.

Angiotensin I is largely converted to angiotensin (1-7) and angiotensin (2-10) by isolated rat glomeruli.

Anti-GBM glomerulonephritis involves IL-1 but is independent of NLRP3/ASC inflammasome-mediated activation of caspase-1.

New enzymatic route for the inactivation of angiotensin.

P304

2119-2132

http://www.w3.org/2001/XMLSchema#string

P31

scholarly article

P356

10.1681/ASN.2016111166

http://www.w3.org/2001/XMLSchema#string

P433

http://www.w3.org/2001/XMLSchema#string

P478

http://www.w3.org/2001/XMLSchema#string

P50

John M. Arthur

Ehtesham Arif

P577

2017-02-15T00:00:00Z

http://www.w3.org/2001/XMLSchema#dateTime

P698

28202497

http://www.w3.org/2001/XMLSchema#string

P932

5491295

http://www.w3.org/2001/XMLSchema#string