The genome-wide transcriptional response to neonatal hyperoxia identifies Ahr as a key regulator.

The genome-wide transcriptional response to neonatal hyperoxia identifies Ahr as a key regulator.

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http://www.wikidata.org/entity/Q38963502
Postnatal Infections and Immunology Affecting Chronic Lung Disease of Prematurity.Transcriptome Analysis of the Preterm Rabbit Lung after Seven Days of Hyperoxic Exposure.Gene Expression Profiling Identifies Cell Proliferation and Inflammation as the Predominant Pathways Regulated by Aryl Hydrocarbon Receptor in Primary Human Fetal Lung Cells Exposed to Hyperoxia.Recent advances in the mechanisms of lung alveolarization and the pathogenesis of bronchopulmonary dysplasia.Looking ahead: where to next for animal models of bronchopulmonary dysplasia?Recurrent copy number variants associated with bronchopulmonary dysplasia.Cardiopulmonary bypass in the newborn: effects of circulatory cell-free hemoglobin and hyperoxia evaluated in a novel rat pup model.Effect of Inhaled Nitric Oxide on Survival Without Bronchopulmonary Dysplasia in Preterm Infants: A Randomized Clinical Trial.
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The genome-wide transcriptional response to neonatal hyperoxia identifies Ahr as a key regulator.

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http://www.wikidata.org/entity/Q38963502
description
2014 nî lūn-bûn
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2014年の論文
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2014年論文
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2014年論文
@zh-hant
2014年論文
@zh-hk
2014年論文
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2014年論文
@zh-tw
2014年论文
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2014年论文
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2014年论文
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name
The genome-wide transcriptiona ...... tifies Ahr as a key regulator.
@en
type
label
The genome-wide transcriptiona ...... tifies Ahr as a key regulator.
@en
prefLabel
The genome-wide transcriptiona ...... tifies Ahr as a key regulator.
@en
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The genome-wide transcriptiona ...... tifies Ahr as a key regulator.
@en
P2093
P2860
P304
P31
P356
10.1152/AJPLUNG.00200.2014
P433
P478
P577
P5875
P698
P921
P932